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COMPREHENSIVE I HUMAN CASE WEEK #7 49-YEAR
OLD PATIENT REASON FOR ENCOUNTER:
INTERMITTENT SQUEEZING CHEST PAIN LATEST
STUDY CASE ACTUAL SCREENSHOTS NEW!!!!! 2025
Exercises Done 68% (of scored items only)
32min 51sec
3 of 5 correct 1 partially correct
(of scored items only)
AA
HistoryNotecard by AA on case
Use this worksheet to organize your thoughts before developing a differential diagnosis list.
- Indicate key symptoms ( Sx ) you have identified from the history. Start with the patient's reason(s) for the encounter and add additional symptoms obtained from further questioning.
- Characterize the attributes of each symptom using "OLDCARTS". Capture the details in the appropriate column and row.
- Review your findings and consider possible diagnoses that may correlate with these
symptoms.(Remember to consider the patient's age and risk factors.) Use your ideas to help the case. guide your physical examination in the next section of
HPI Sx = chestpain
Sx
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Sx
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Sx
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Sx
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Sx
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Onset 2.5 hours ago
Location deepin chest, pierces
through back
Duration
Characteristics worsens with deep inspiration
Aggravating worsens when laying
down, moving, and breathing
Relieving eased pain when leaning
forward
Timing /
Treatments
Severity 8/
COMPREHENSIVE I HUMAN CASE WEEK #7 49-YEAR
OLD PATIENT REASON FOR ENCOUNTER:
INTERMITTENT SQUEEZING CHEST PAIN LATEST
STUDY CASE ACTUAL SCREENSHOTS
History of Present Illness
Category Data entered by AA
Reason for Encounter Chest pain
History of present illness A 57 - year-old female 10 days status post inferolateral STEMI with stent placement, presents to the ED
with a 2.5-hour history of progressively worsening
chest pain. The pain is sharp and stabbing, gets
to pierce through her back with inspiration. Pain
is partially relieved by sitting up and leaning forward.
She reports mild nausea without vomiting, denies fever/chills,
palpitations, lightheadedness/syncope, SOB, cough, URI, or
extremity/catheterization site pain or swelling. She reports
compliance with her antiplatelet medications (ASA
and clopidogrel), pantoprazole, and aspirin since
discharge. On exam, patient is febrile, shallow
respirations at normal rate, low-normal oxygen
saturation, and has pericardial friction rub. PMH
is significant for hypertension, hyperlipidemia, tobacco
abuse, obesity, and type- 2 diabetes.
family history of heart attack and
Past Medical History
Category Data entered by AA
COMPREHENSIVE I HUMAN CASE WEEK #7 49-YEAR
OLD PATIENT REASON FOR ENCOUNTER:
INTERMITTENT SQUEEZING CHEST PAIN LATEST
STUDY CASE ACTUAL SCREENSHOTS
Category Data entered by AA
General Slightly fatigued. Laying still to avoid movement, breathingslowly.
Integumentary / Breast Warm skin; no significant diaphoresis.
HEENT / Neck Denies any problems with headaches, double vision, difficulty with night vision, hearing problems, ear pain, sinus
problems, chronic sore throats, or difficulty swallowing.
Cardiovascular Chest pain. Heart attack 10 days ago.
Denies history of irregular heartbeats nor Hypertension Hyperlipidemia
palpitations.
Respiratory Denies wheezing and sputum production.
Gastrointestinal Reports mild nausea and reflux.
Genitourinary Denies urinary frequency, pain, incontinence, or difficulty.
Musculoskeletal Denies chest injury. No problems with muscles and joints.
Allergic / Immunologic
Endocrine Denies problems with heat or cold intolerance, increasedthirst,
increasedsweating, frequent urination,or change in appetite.
Hematologic / Lymphatic No reports of bruising, bleeding gums, sites of increased bleeding.
nose bleeds, or other
Neurologic Denies dizziness, seizures, numbness, or weakness.
Psychiatric Reports nervousness due to chest pain.
Physical Exams
Category
Data entered by AA
General Obese with a BMI of 29.4.
AOx Skin is dry and febrile. Skin Skin warm and dry with no lesions.
Nails without ridging, pitting, or peeling.
Capillary refill < 2 sec. Quincke’sTest: Blanchingobserved.
The Patient is Alert on AVPU Score.
The pupils are rounded, central, responsive to light & equal on both sides. Exposure:
No apparent injuries in body.
General Examination
Patient is alert , conscious , oriented to time , place and persons , average built , quiet facial
expression , no special decubitus , average intelligence ,he's cooperative
Cardiac Examination
By inspection:
There is no skin redness, pigmentation, ulceration, fistulae or scars.
Respiration is abdomenothoracic.
No visible pulsation at the apex of the heart.
By Palpation:
Palpable pulsation of the heart at the fifth intercostal space in the midclavicular line.
There are no masses, swellings, tenderness or pulsating masses.
By Auscultation:
No abnormal cardiac sounds, muffling or murmurs were heard at the apex of the heart,
xiphisternal area and aortic areas.
Differential Diagnosis
- Acute Coronary Syndrome (most probable)
- Pericarditis excluded by absence of localized friction rub over pericardium.
- Aortic Dissection excluded by absence of unequal pulsation or pulsating masses.
- Musculoskeletal Pain excluded by not being aggravated by chest movement and not being
relieved by changing posture.
- Pleurisy, Bronchitis, Broncho-Pneumonia or Lung Carcinoma excluded by absence of
cough, haemoptysis, friction rub on the chest and audible abnormal breathing sounds.
- Esophagitis or peptic ulcer excluded by absence of history of dysphagia, odynophagia,
hematemesis or heartburn.
Provisional Diagnosis
Pulmonary embolism Sudden onset, pleuritic pain, and dyspnea
lOMoARcP SD| 46 020670
Aortic dissection Tearing pain with radiation to back, neurologic symptoms
Pericarditis Positional ache, dyspnea
Pneumothorax Pleuritic pain and dyspnea
Acute coronary syndrome Vague, pressure-like pain, radiation to arm, neck, jaw
Esophageal rupture Constant retrosternal, epigastric pain, history of inciting event
Pneumonia Pleuritic pain, cough, dyspnea, chills
SPECIFIC CAUSES OF CHEST PAIN Angina Pectoris
The pain of chronic stable angina is episodic and lasts 5 to 15 minutes. It is precipitated by exertion and
relieved with rest or sublingual nitroglycerin within 3 minutes. The pain is typically visceral in nature
(aching, pressure, and squeezing), with radiation to the neck, jaw, arm, or hand. In individual patients
the character of each attack varies little with recurrent episodes. Most patients can differentiate their
usual angina from other causes of pain. Physicians evaluating patients with stable angina should screen
carefully for changes in the pattern that would suggest a shift from stable to unstable angina or even
suggest a different diagnosis.
Unstable Angina :
Patients who complain of recent onset of angina, changes in the character of the angina, or angina at
rest are thought to have an unstable pattern of their angina. They are at risk for an AMI or sudden
cardiac death (see Chapter 21 for management).
Variant (Prinzmetal) Angina
This form of angina is thought to be due to spasm of the epicardial vessels in patients with normal coronary
arteries (one third of cases) or in patients with underlying atherosclerotic disease (two thirds of cases). Pain
typically occurs at rest and may be precipitated by the use of tobacco or cocaine. The ECG typically shows
ST- segment elevations during an acute attack. Acute Myocardial Infarction
Ischemic pain that lasts longer than 15 minutes, is not relieved by nitroglycerin, or is accompanied by diaphoresis,
dyspnea, nausea, or vomiting suggests the diagnosis of AMI. The clinician must understand the limitations of the
screening tools used in the ED and should have a high level of suspicion for AMI in patients with risk factors and
prolonged or persistent symptoms for whom there is no other clear diagnosis. Aortic Dissection
This diagnosis should be suspected in the patient who complains of sudden onset of severe, tearing pain in the
retrosternal or midscapular area. High-risk patients are also those at risk for AMI, specifically the middle-age
hypertensive male. The patient may be hypertensive or hypotensive in shock. There may be a diastolic murmur of
aortic regurgitation, indicating a proximal dissection, or distal pulse deficits, indicating a distal dissection. The
dissection may occlude coronary ostia, resulting in myocardial infarction, or the carotids, resulting in cerebral
ischemia and stroke. Chest x-ray, computed tomography, trans esophageal echocardiography (TEE), and
angiography can aid in the diagnosis of this condition. Musculoskeletal Causes
Chest pain due to irritation or inflammation of structures in the chest wall is commonly seen in the ED. Possible
causes include costochondritis, intercostal strain due to severe coughing, and pectoralis muscle strain in the
setting of recent physical exertion. Patients will complain of sharp pain that is worsened with movement of the
chest wall (eg, coughing, and some pain that can be elicited by palpation of the chest wall). These findings in
patients without any other symptoms and no history of significant cardiac disease support the diagnosis of
musculoskeletal pain. This pain is generally responsive to no steroidal anti-inflammatory drugs. It is important to
emphasize that the presence of chest wall tenderness does not rule out the possibility of myocardial ischemia.
lOMoARcP SD| 46 020670
Gastrointestinal Causes
Esophageal reflux, dyspepsia syndromes, and esophageal motility disorders can produce chest pain that is difficult
to distinguish from ischemic pain. Patients may complain of burning, gnawing pain associated with an acid taste
radiating into the throat. Pain may be exacerbated by meals, worsen when supine, and may be associated with
belching. Clinicians should determine whether the symptoms are due to a GI disorder based on the clinical
presentation and the absence of findings and/or risk factors suggesting an ischemic cause. Diagnostic decisions
should not be made on the basis of a response to a therapeutic trial of antacids, GI cocktails, or nitroglycerin.
EMERGENCY DEPARTMENT CARE AND DISPOSITION
Assume that every patient complaining of chest pain might be having an AMI.
Patients with suspicious histories should have large-bore IV line established, a cardiac monitor,
supplemental O 2 , and ECG obtained as soon as possible. Vital signs and pulse oximetry should be
monitored continuously.
Ask patients about cardiac risk factors, preexisting coronary artery disease, quality of chest pain, time
of onset and duration of symptoms, and whether the pattern has been stable, unstable, continuous, or
intermittent. Ask specifically for clues to noncardiac causes of chest pain: ability to elicit pain by
movement or cough; the relation of pain to meals; or pain that is of sudden onset, referred to the back,
or pleuritic in nature.
Examine patients while noting evidence of heart failure or valvular insufficiency, pericardial rubs, or
tenderness of the chest wall. Specifically, physicians should ask whether pain elicited on palpation of
the chest wall exactly reproduces the patient's pain.
An ECG should be obtained on all patients for whom there is a reasonable suspicion of myocardial
ischemia. A normal ECG, although minimizing the likelihood of an AMI, does not definitively rule out the
possibility of MI.
If the etiology of chest pain remains unclear in some patients, clinicians should consider more
diagnostic tests as guided by clinical suspicion and findings.
Clinicians should not use patients' clinical response to GI cocktails, nitroglycerin, or NSAIDs to exclude
the possibility of myocardial ischemia.
In patients with nondiagnostic ECGs for whom there is a clinical suspicion for ischemia, clinicians should
consider provocative testing, echocardiography, or admission and observation. Physicians should not
rely on serum enzyme testing to rule out the possibility of clinically significant disease.