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This comprehensive document provides a detailed guide for nurses on various gastrointestinal and renal disorders, including their symptoms, diagnosis, treatment, and patient management. Topics covered include ibs, appendicitis, post-cholecystectomy syndrome, urinary tract infections, urolithiasis, kidney trauma, benign prostatic hyperplasia, and more. The guide also includes patient teaching points and key nursing considerations.
Typology: Study Guides, Projects, Research
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Diarrhea:
Normal stomach- food is swallowed --> peristalsis occurs to squeeze food into the stomach. LES relaxes, food enters, LES closes In GERD- impaired esophageal motility
Differentiate between heartburn and cardiac chest pain
Sleep with HOB elevated Avoid straining Avoid vigorous exercise
What is it? Gastritis is an inflammation of the gastric mucosa, may be acute or chronic. Acute: thickened, reddened mucosa with prominent rugae or folds. Varying degrees of mucosal necrosis and inflammatory reaction. Complete healing usually occurs in within a few days. If stomach is not involved complete recovery usually occurs with no gastric inflammatory reaction. If muscle is affected, hemorrhage may occur during acute. Chronic gastritis: patchy, diffuse inflammation of the
Results in pernicious anemia (a condition in which not enough red blood cells are produced due to deficiency of vitamin B in the body) Associated with an increased risk for gastric cancer. Type A: Nonerosive Type B: Erosive Atrophic Inflammation of the glands and the fundus and body of the stomach Associated with the presence of antibodies to parietal cells and intrinsic factors – autoimmune cause likely. Long term NSAID use ETOH, coffee, caffeine, and corticosteroids Accidental ingestion of erosive chemicals Effects the glands of the antrum but may involve the entire stomach. Caused H.Pylori infection Smoking, ETOH abuse, radiation therapy Pyloroplasty Chrons disease Uremia Type of chronic gastritis that effects the older population Exposure to toxic substances (lead, nickel) in the workplace H.Plyori Autoimmune disorders Associated with gastric cancer and MALT Signs and symptoms: Acute Chronic Rapid onset of pain Pain relieved by food Nausea and vomiting Anorexia Hematemesis Nausea and vomiting Gastric hemorrhage Intolerance to fatty/spicy foods Heartburn Pernicious anemia Anorexia Prevention/patient teaching: Well-balanced diet Avoid ETOH Caution
aspirin, NSAIDS Avoid caffeine
Therapeutic management: Determine cause Diagnostic tests: EGD, Abd XRAY, Abd/pelvic CT Labs: CBC - Hemoglobin: 14-18 M 12-16 F Hematocrit: 42-52% M 37-47% F White Blood Cell: 5- 10mm RBC: 4.7- 6.1mm 4.2-5.4mm Platelet Count: 150- 400mm Liver panel Lipase- 0- 110 IU/L Medications: Symptom manageme nt- Antiemetics Analgesics H2 Receptor Blockers – Famotidine Sulfcrate Antacid
s PPIs
What is it? Functional GI disorder causes chronic or recurrent diarrhea, constipation and LLQ abdominal pain and bloating. Changes in motility and increased or decreased bowel transit times results in changes in normal bowel patterns: IBS- D – diarrhea IBS- C – constipation IBS- A – alternating IBS-M – mix Symptoms appear in young adulthood and continue throughout the patient's life. Etiology & Cause: Unclear etiology Environmental – caffeine, carbonation, dairy, infectious agents. Immunologic, genetic – cytokine genes, IL-6, IL-8, tumor necrosis factor- alpha. Risk factor: Women 2 times more than likely than men (possible hormone related) Mental health disorders – anxiety, depression Pain and disease may lead to secondary mental health disorders Assessment:
Ask patient history -
Bulk forming laxative- Metamucil Antidiarrheals – Imodium, psyllium
What is it? Weakness in the abdominal muscle wall through which a segment of the bowel or other abdominal structure protrudes. Can also penetrate through other defect in the abdominal wall, diaphragm, and abdominal cavity. Development of a hernia are congenital or acquired muscle weakness and increased intra-abdominal pressure