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The challenges and changes in wound healing and skin health that occur in older adults. It covers topics such as the increased time for skin renewal, the redistribution of fat leading to exposed bony surfaces, the fragility of blood vessels and skin, the development of secondary lesions, and the impact of comorbidities on wound healing after burn injuries. The document also addresses the assessment and management of conditions like cellulitis, herpes zoster, and skin cancer in the elderly population. Additionally, it covers the screening, diagnosis, and treatment of abdominal aortic aneurysm (aaa), a serious cardiovascular condition that is more common in older adults. Valuable insights into the unique skin and wound care needs of the aging population, which is essential for healthcare professionals working with this demographic.
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Skin renewal turnover time increases to approximately 87 days in older adults, compared with 20 days during youth.
The perceived extended healing time is not related to diet.
This is false hope, as there is no medication that will heal this wound quickly.
Prophylactic antibiotics are not appropriate when there are no signs or symptoms of infection.
Lack of activity alone does not cause skin breakdown.
Fat is redistributed to the abdomen and thighs, leaving bony surfaces, such as the face, hands, and sacrum, exposed to potential injury, especially skin tears from shearing, friction forces and pressure ulcer development.
Although losing weight may be a risk factor for falling, it is not directly related to skin breakdown.
There is no evidence that she is picking at herself, as there is nothing reported anywhere else on her arms.
Markings on the skin may be signs of aging, a disease, or maltreatment.
Poorly healing wounds or chronic pressure ulcers may signal a problem not only with the patient but with the caregiver's ability to provide adequate care. Welts, lacerations, burns, and distinctive markings may indicate a need for intervention.
This is a result of the nurse practitioner addressing it further rather than the reason for addressing it.
A professional cannot assume abuse without good reason.
Secondary lesions (infections) arise from changes to the primary lesion.
Secondary lesions are not necessarily the result of an underlying disease.
Secondary lesions can be treated with medications or surgery.
Secondary lesions arise as a condition not normal to aging.
An injury would not stimulate growth.
A reaction to a detergent would more likely be a rash.
Lesions that warrant biopsy are those that have changed, bleed, or are painful.
The ability to put on her ring is not the problem.
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First-degree burns involving the epidermis are erythematous and painful but do not blister.
Second-degree burns involve the dermis and are characterized by blisters.
The patient presents with erythematous skin, painful with blisters, which indicates both first- and second-degree burn areas.
In third-degree burns there is no sensation when the wound is pinpricked.
Primary prevention includes educational programs designed to educate the public on safety. For example, the individual smoking in bed would hopefully benefit from smoking cessation programs in the community, as well as instruction in safety precautions.
Threatening refusal of care is not ethical.
The patient is at risk, not the family.
The fact that the patient smokes is not the issue; safety is the issue.
As one ages, there are significant changes in the skin, which becomes thinner, providing a less effective barrier to external stimuli.
With aging, there are fewer appendages and decreased vascularity.
Thinner skin and diminished nerve function often result in a higher incidence of deeper burns.
Advanced age results in a weakened immune system.
Along with the burden of various comorbidities, the fragility of older skin leads to delayed wound healing and reepithelialization after burn injury.
Deep dermal burns extend further into the dermis; third-degree burns involve the full dermis, extending into the subcutaneous tissue.
In these burns there is pain from exposed nerve endings, but by the second day, pain is often described more as pressure.
The first step in treatment is to stop the burn.
Destroyed nerves do not register pain.
Superficial dermal burns involve the dermis and are characterized by blisters. The underlying tissue is pink, moist, and hypersensitive to touch.
After administration of appropriate pain medication, wound management can begin.
Burn wounds should be immediately doused in cool tap water to disperse any remaining heat in the tissue.
Detergents and antibacterial soaps are not indicated. Burn wounds should be cleaned with mild soap and rinsed.
For small surface area burns, it is good to remove any loose tissue during cleansing and allow intact blisters to remain.
First-degree burns do not exhibit blisters, and third-degree burns do not exhibit pain.
Influenza is systemic and not localized in any one area.
The patient has no respiratory symptoms.
Signs of cellulitis include worsening of erythema, edema, tenderness, and pain that has occurred for a few days. Symptoms are usually sudden. Systemic symptoms which indicate serious toxicity include fever, hypotension, and tachycardia.
Necrotizing fasciitis exhibits diffuse swelling of an arm or leg with bullae.
Systemic symptoms that indicate serious toxicity include fever, hypotension, tachycardia, leucocytosis, lymphadenopathy, and lymphangitis.
Oral antibiotics are sufficient for mild cellulitis and IV antibiotics for organisms such as MRSA.
There are several drugs effective with cellulitis; dicloxacillin is one of them.
Treatment of MRSA should be guided by wound culture results, but not cellulitis.
The drug of choice is typically given for 7 days.
If the wound is grossly contaminated and the patient's last tetanus booster was 5 to 10 years ago, the practitioner should consider giving another booster at this time.
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Patients usually experience itching, burning, or tingling pain at the site 4 to 5 days before the eruption appears.
Although there are general systemic symptoms, there is also itching, burning, and pain in the waist, which is leading to a herpes zoster diagnosis.
The patient has initial contact with VZV in the form of chicken pox. Individuals who are immunosuppressed are more likely to develop herpes zoster.
Herpes zoster is characterized by pain along the dermatomes and vesicles, which is not symptomatic of influenza.
This infection is most common in adults over 55 years old. The risk of herpes zoster increases with age.
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Signs of malignancy include elevation; the original lesion may also have enlarged in size.
Common locations for skin cancers are the scalp, ears, lower lip, and dorsal side of the hands.
Signs of malignancy include inflammation of the lesion.
The incidence of all types of skin cancers increases with age and the degree and intensity of sun exposure.
Certain genetic predispositions can contribute to the development of skin cancer, and there is a familial tendency to develop melanoma.
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The key to prevention of recurrence is to keep the area dry. Use a hairdryer to thoroughly dry the area after bathing.
The key to prevention of all types is to keep the skin cool and dry.
The use of aluminum acetate solution (Burow's) and the application of antifungal or absorbent powder have all been shown to prevent recurrence.
Avoiding occlusive footwear, wearing absorbent materials, and practicing good hygiene offer the best primary prevention.
Newer agents are more likely to cure tinea pedis than the older generation of antifungals, including clotrimazole, which is fungistatic, whereas terbinafine is fungicidal.
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The clinician should begin with auscultation of the carotid arteries bilaterally because cardiac murmurs will usually radiate into the carotid arteries.
After auscultation of the carotid and subclavian arteries, the clinician should proceed to palpation of the brachial, radial, and ulnar arteries.
After auscultation and palpation, careful inspection should be performed of the distal fingers and the nail beds.
Diagnostic testing is usually ordered when areas of concern are found.
AAA presents the signal symptoms of persistent or intermittent pain in the middle or lower abdomen, radiating to the lower back.
Venous leg ulcers present signal symptoms of swelling that subside with elevation of lower extremities, eczematous skin changes, dull ache in lower extremities, and presence of varicosities.
PVD is a disease that alters blood flow to or from the extremities and vital organs other than the heart. It presents signal symptoms of pain, intermittent claudication of the feet, and tissue loss in affected leg/arm.
Lymphedema presents the signal symptoms of swelling of the affected body part, usually the limb, because of impaired flow of lymph fluid.
CT screening is indicated when surgery is planned.
CBC may be a secondary screening when surgery is planned.
Ultrasound in the abdominal area is the best initial screening test for AAA.
Angiography screening is indicated when surgery is planned.
A reading of 100 to 120 mm Hg is too high.
A reading of 90 to 120 mm Hg is too high.
Good arterial pressure for those with AAA should fall between 60 and 70 mm Hg.
A reading of 80 to 100 mm Hg is too high.
A vascular surgeon is a specialist who is highly trained to treat diseases of the vascular system.
A neurosurgeon is a physician who specializes in the diagnosis and surgical treatment of disorders of the central and peripheral nervous system.
A cardiologist is a doctor who specializes in the study or treatment of heart diseases and heart abnormalities.
Internists are specialists who apply scientific knowledge and clinical expertise to the diagnosis, treatment, and compassionate care of adults across the spectrum, from health to complex illness.
Gynecological cancer is a contributing factor for secondary lymphedema, but it is not the best option for this scenario because of Anne's history.
Breast cancer is the best option and is a contributing factor for secondary lymphedema in Anne's case because of the previous mastectomy.
Urological cancer is a contributing factor for secondary lymphedema, but it is not the best option for this scenario because of Anne's history.
Infection is a contributing factor for secondary lymphedema, but it is not the best option for this scenario because of Anne's history.
Smoking remains the most important risk factor to PVD.
Hypertension is an associated risk factor to PVD.
A strong family history of the disease is an associated risk factor to PVD.
Hypercoagulopathy is an associated risk factor to PVD.
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Marfan syndrome is a genetic disorder that affects the body's connective tissue. It is not related to the diagnosis of PAD.
Atelectasis is a condition where some, or all, of the air-filled sacs (alveoli) inside the lungs collapse, thereby reducing the lungs' capacity to deliver oxygen to the body. It is not related to the diagnosis of PAD.
Raynaud's phenomenon is a type of vascular disease characterized by a pale to blue to red sequence of color changes of the digits, most commonly after exposure to cold, and should be considered with the diagnosis of PAD. Buerger's disease is also a differential diagnosis for this disease.
Carpal tunnel syndrome is a common condition that causes pain, numbness, and tingling in the hand and arm. It is not related to the diagnosis of PAD.
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Education about hypertension management is important for patients with AAA. Antihypertensive agents are used to reduce tension on the vessel wall in patients with AAAs who have elevated blood pressure.
Patients with AAA need to learn about control of hypercholesterolemia (also called high cholesterol). Hypercholesterolemia is characterized by high serum cholesterol levels and premature coronary atherosclerosis. Hypercholesterolemia is one of the factors promoting the arteriosclerotic process and is a major cause of aortic aneurysm.
Smoking cessation should be considered by patients with AAA. Smoking appears to increase the risk of aortic aneurysms. Smoking can be damaging to the aorta and weaken the aorta's walls.
Chondromalacia is a pain in the knee and has no relationship to AAA.
Pain is the most common symptom of an AAA. Pain associated with an AAA may be in the abdomen, chest, lower back, or groin area. The pain may be severe or dull. Sudden, severe pain in the back or abdomen may mean the aneurysm is about to rupture.
There is no dominant ethnic group that develops AAA, but there is a familial history associated with AAA development.
There is no dominant ethnic group that develops AAA.
Onset occurs around age 50 years for men and 60 years for women. Incidence steadily increases with age and peaks at age 80 years. AAA is five times more likely in men than in women.
There is no dominant ethnic group that develops AAA, but there is a familial history associated with AAA development.
According to studies, AAAs are the 13th leading cause of death in the United States. Mortality rates for ruptured aneurysms are 70% to 90% compared with 5% operative mortality for elective open surgical repair, and 2% to 3% for endovascular stent AAA exclusion.
mean arterial pressure ranges? Select all that apply.
Nicardipine is an IV antihypertensive agent that should be used to rapidly and consistently maintain blood pressure in mean arterial pressure ranges.
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Esmolol is an IV antihypertensive agent that should be used to rapidly and consistently maintain blood pressure in mean arterial pressure ranges.
Nitroglycerin is an IV antihypertensive agent that should be used to rapidly and consistently maintain blood pressure in mean arterial pressure ranges.
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