Cellular responses to stress, Schemi e mappe concettuali di Patologia Generale

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morphologic. halimarks ; ; lized chandes lead to lead 16 "ossa TT membrane integrity loss |—aT® associfited blebbing adaptations 2 protein synthesis defects bosomes ni detachment when the cell is increased decreased cell type no longer able to gell size increased cell size replacement cell number and number adapt cytoskeletal damage DNA damage chromatin clumping hypertrophy hyperplasia atrophy metaplasia pathologic/physiologic pathologic/physiologic physiologic Pupo increased ) ( stimulation ) ( compensatory ) ( excessive during ‘stem cells undifferentiated functional | | by growth increase action of normal | pathologic | reprogramming || Mesenchymal demand factors needed hormones | | development ces < workload innervation loss depletion of ATP mitochondrial membrane di damage permeability inadequate ) | endocrine nutrition | | stimulation loss leads to necrosis < blood ‘supply permeability transition pore apoptosis DNA damage