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Hemodynamic - Medical Science - Lecture Notes, Study notes of Biology

AMET UniversityBiology

Hemodynamic, Superficial Hemorrhage, Heart Failure Cells, Nutmeg Liver, Lines of Zahn, Disseminated Intravascular Coagulopathy, Disseminated Intravascular Coagulopathy, Trousseau Syndrome are some points from this lecture. In my uploads you will find many files explaining one or more basic term of Medical Science. This is introductory course for Medical Science students.

Typology: Study notes

2011/2012

Uploaded on 12/21/2012

ekmbr
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HEMODYNAMIC
HEMORRHAGE:
• PURPURA: Diffuse superficial hemorrhage, up to 1cm in diameter.
• ECCHYMOSIS: Bruise.
• PETECHIA: Pinpoint hemorrhage, esp. in conjunctivae and skin.
HYPEREMIA
• PHYSIOLOGIC (ACTIVE): Exercise
• PASSIVE: Congestive
o Lung: Heart Failure Cells have hemosiderin.
o Liver: Nutmeg Liver
o Spleen
o Edema and Ascites
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HEMODYNAMIC

HEMORRHAGE:

  • • PURPURA:ECCHYMOSIS: Diffuse superficial hemorrhage, up to 1cm in diameter. Bruise.
  • PETECHIA: Pinpoint hemorrhage, esp. in conjunctivae and skin. HYPEREMIA
  • • PHYSIOLOGIC (ACTIVE): ExercisePASSIVE: Congestive o o Lung:Liver: Heart Failure CellsNutmeg Liver have hemosiderin. o o SpleenEdema and Ascites

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THROMBOSIS: Clot. Technically a thrombus is adherent to vascular endothelium.

  • LINES OF ZAHN:mortem and separates into layers. Help distinguish a thrombus from a post-mortem clot which is where blood coagulates post-

EMBOLISM: Anything that knocks loose and lodges in the blood.

  • • AIR EMBOLUS: As inAMNIOTIC FLUID EMBOLUS: Can lead to Decompression Disease Disseminated Intravascular Coagulopathy (DIC), due to the
  • presence ofBONE MARROW EMBOLUS^ Thromboplastin^ in amniotic fluid.
  • • FAT EMBOLUS: Large bone fracturesFOREIGN BODY EMBOLUS: Bullet
  • PARADOXICAL EMBOLUS:patent foramen ovale or other septal defect. Emboli originally from venous side go to arterial side and cause an infarct, due to a
  • THROMBO EMBOLUS: Most common. ANASARCA: Profound, generalized edema. DISSEMINATED INTRAVASCULAR COAGULOPATHY (DIC):which consume all platelets and result, paradoxically, in uncontrolled bleeding in certain areas. Usually fatal. Thousands of intravascular micro-clots
  • CAUSES: o Amniotic Fluid Embolus, release of Thromboplastin into blood. o Gram negative endotoxic shock TROUSSEAU SYNDROME:cancer Paraneoplastic syndrome of hypercoagulable state. Classically associated with pancreatic

INFARCTION: Acutely, it leads to Coagulative Necrosis.

  • • RED INFARCT:PALE INFARCT: Pulmonary infarct, or infarct of an organ that has dual blood supply. Single blood supply, as in kidney.

EDEMA: OK? SHOCK:

  • • CARDIOGENIC SHOCKSEPTIC SHOCK
  • TOXIC SHOCK: Due torare phage. Staph and Strep exotoxin, TSS. These bugs are induced to make TSS by the presence of a
  • • HYPOVOLEMIC SHOCKNEUROGENIC SHOCK: ANS failure.
  • STAGES: o REVERSIBLE: High vasoconstriction, vascular tone. o o CRITICAL: Very high TPRIRREVERSIBLE: Pooling and stagnation, interstitial edema, severe acidosis.

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