Memory Disorders - Memory and Amnesia - Lecture Slides, Slides of Human Memory

Memory Disorders, Influences on Memory, Kinds of Memory Disorders, Alcohol and Memory, Alcoholic Amnesia, Alcoholic Blackout, Sleep and Memory, Sources of Organic Dysfunction are some points from lecture of Memory and Amnesia.

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PSY 335
Memory and Amnesia
Memory Disorders
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PSY 335

Memory and Amnesia

Memory Disorders

Midterm Results

Score Grade N 51 - 60 A 4 45 - 50 B 13 39 - 44 C 13 33 - 38 D 7 0 - 32 F 3 Top score = 59, Top score for curve-setting = 57

Kinds of Memory Disorders

 Organic – having a physical cause

 Functional – having a psychological cause

 Dys (as a prefix) means difficulty or limited ability to perform.

 A (as a prefix) means complete inability or lack of a function.

Alcohol & Memory

 Alcoholic amnesia – alcohol prevents consolidation so nothing is remembered and no memory can be recovered.

 Alcoholic blackout – state-dependent memory, so recall is possible if one is back in the same state.

 Because many crimes are committed while drunk, memory failure is frequently blamed on alcohol.

Sources of Organic Dysfunction

 Accident

  • Car accidents and other injuries (e.g., N.A.)
  • War

 Disease

  • Encephalitis (viral) – inflammation of the lining of the brain, causing swelling.
  • Stroke
  • Alzheimer’s disease
  • Korsakov’s syndrome (prolonged alcoholism)

Alzheimer’s Disease

 A fatal degenerative disease caused by cell failure – neurofibrillary tangles and plaques that interfere with cell function.

  • All areas of the brain are eventually affected, but frontal lobes and memory go first.

 Confusions and memory problems do not resemble normal aging, amnesia or other memory problems.

Classification of Disorders

 See Parkin, Ch 5, for tests used to assess memory problems.

 Disorders classified by type of symptom:

  • Generalizing – confusion, fuzziness, mental slowing.
  • Localizing – few generalizing symptoms but impairment of specific functions.

 Clusters of symptoms are a syndrome.

  • Concern about symptoms is a symptom itself.

Frontal Lobe Deficits

 Confabulation – production of a false memory.

  • Momentary confabulation – responses that could be correct.
  • Fantastic confabulation – responses clearly fictional.

 Source amnesia – fact is remembered but not the source.

 Memory of temporal order.

Frontal Lobe Deficits (Cont.)

 Faulty encoding and poor representation may be a cause of poorly focused search.

  • Information is needed to guide search.

 The left frontal lobe guides encoding.

 The right frontal lobe guides retrieval.

Frontal Lobe Deficits (Cont.)

 Emotional deficits:

  • Cognitive apathy, lack of motivation
  • Flattened affect

 Impaired awareness of memory loss:

  • Inaccurate assessment of performance
  • Lack of distress

 If confabulations are believed by others, no feedback on normalcy.

Peter Sellars in “Dr. Strangelove: or How I learned to story worrying and love the bomb”

Damage to the Parietal

Association Cortex

 Confusion about directions, inability to use words describing spatial relations:

  • Under, up, down  Inability to name body parts or point to parts of the body.

 Capgras syndrome (rt. Posterior parietal) inability to recognize close family members

  • Sometimes animals or even furniture
  • Invasion of the body snatchers

Pure Word Deafness

 A person can hear and speak, read and write normally but cannot understand speech.

 Occurs with bilateral destruction of the auditory cortex or disconnection from Wernicke’s area.

 Because Wernicke’s area is not damaged, speech produced is OK.

Perceptual Deficits

 Aphasia – involves inability to name something.

 Agnosia – involves inability to recognize something.

 Visual agnosias – inability to combine individual visual impressions into complete patterns.