NR667 Chamberlain CEA Week 8 Exit Exam Test Questions with Answers, Exams of Nursing

NR667 Chamberlain CEA Week 8 Exit Exam Test Questions with Answers

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NR667 Chamberlain CEA Week 8 Exit Exam Test Questions with Answers
1.
Blood Flow
Lungs ® Pulmonary Veins ® Left
Atrium
®
Aorta
®
Body
Tissues
®
Vena
Cava
®
Right
Atrium
®
Right
Ventricle
®
Pulmonary
Arteries
®
Lungs
2.
Symptoms
with
right
vs
left
side
heart
blockage
· Blockage on the left side of the
heart backs up and causes lung
symptoms
· Blockage on the right side of the
heart backs up and causes body
symptoms (peripheral edema)
3.
HNC8 HTN Guidelines
Defined as 140/90
Treatment
algorithm:
Less than 60 years old - 140/90
>
60
years
old
-
Defined
as
150/90
(more
leniency
b/c
we
do
not
want
to drop their BP too low)
4.
What hypertension medication should someone
ACE or ARB (protects
kidneys)
with DM and/or CKD be on?
5.
What HTN medication should an African Ameri-
can pt be on?
6.
What HTN medications should be used in pa-
tients with heart failure?
CCB
Carvediolol
and
Thiazide
diuretics
7.
Common side effects from ACE inhibitors
cough,
angioedema
8.
What HTN medication is contraindicated if an
ACE inhibitor caused angioedema?
ARB
pf3
pf4
pf5
pf8
pf9
pfa
pfd
pfe
pff
pf12
pf13
pf14
pf15
pf16
pf17
pf18
pf19
pf1a
pf1b
pf1c
pf1d
pf1e
pf1f
pf20
pf21
pf22
pf23
pf24
pf25
pf26
pf27
pf28

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NR667 Chamberlain CEA Week 8 Exit Exam Test Questions with Answers

  1. Blood Flow Lungs ® Pulmonary Veins ® Left

Atrium ® Aorta ® Body Tissues ® Vena Cava ® Right Atrium ® Right Ventricle ® Pulmonary Arteries ® Lungs

  1. Symptoms with right vs left side heart blockage · Blockage on the left side of the

heart backs up and causes lung symptoms · Blockage on the right side of the heart backs up and causes body symptoms (peripheral edema)

  1. HNC8 HTN Guidelines Defined as 140/

Treatment algorithm: Less than 60 years old - 140/ > 60 years old - Defined as 150/ (more leniency b/c we do not want to drop their BP too low)

  1. What hypertension medication should someone ACE or ARB (protects kidneys)

with DM and/or CKD be on?

  1. What HTN medication should an African Ameri-

can pt be on?

  1. What HTN medications should be used in pa-

tients with heart failure?

CCB

Carvediolol and Thiazide diuretics

  1. Common side effects from ACE inhibitors cough, angioedema
  2. What HTN medication is contraindicated if an

ACE inhibitor caused angioedema?

ARB

NR667 Chamberlain CEA Week 8 Exit Exam Test Questions with Answers

  1. What HTN medication should a heart failure pt

NEVER be on?

CCB

(These cause the heart to "relax" which is not good in HF pts)

  1. 2 types of CCBs Dihydropyridines & Non-dihy-

dropyridines

  1. What are dihydropyridine CCBs used for? BP control
  2. Example of a dihydropyridine CCB and side ef-

fects Amlodipine Bradycardic side ettects, peripheral edema, constipation

  1. What are non-dihydropyridine CCBs used for? arrhythmias
  2. Example of a non-dihydropyridine CCB and side cardizem

effects

  1. The atria (top chambers of the heart) work on

which electrolytes?

  1. The ventricles (bottom chambers of the heart)

work on which electrolytes?

  1. Conditions in the atria needs medications that

work on K+ or Ca such as ..

  1. Conditions in the ventricles needs medications

that work on K+ or Na such as .. Tachycardic side ettects/palpita- tions - these meds were peripher- ally and have a rebound tachycar- dia K+ (potassium) and Ca (calcium) Na (sodium) and K+ (potassium) Cardizem (CCB) or Amiodarone (potassium channel blocker) Amiodarone (potassium channel blocker)

Which structural heart condition can cause syn- cope or near-syncope?

  1. Which structural heart condition cause a harsh, high-pitches sound that can be heard in the neck or on the right side of the chest near the 2nd intercostal space? Aortic regurgitation/insuflciency
  2. Which structural heart condition is very loud and Mitral regurgitation/insuflciency can be heard on the lower left side of the chest?
  3. What are the 2 most common places for a AAA? infra-renal and ascending aorta
  4. Which aortic aneurysm requires surgery right away? Stanford A (ascending)
  5. Which aortic aneurysm is often treated medical- Stanford B (descending) ly or with a possible graft (but does not often need surgery)?
  6. What is a medical intervention that should be done for a patient with a Stanford B aneurysm?
  7. What class medication should NEVER be given to a patient with an aneurysm or any sort of connective tissue disorder?
  8. What are the 4 fat-soluble vitamins? (stay in the body for a long time)
  9. What percentage of pulmonary emboli or DVTs are provoked? Keep BP low flouroquinolones (end in "floxicin") ADEK 70%
  10. at least 3 months

How long should a patient with a provoked PE or DVT be treated with an anticoagulant?

  1. How long should a patient with a non-provoked at least 3 months, but could be life- PE or DVT be treated with an anticoagulant? long if any recurrence
  2. What is the Virchow's triad? 3 broad categories of factors that are thought to contribute to throm- bosis
  3. What are things that could contribute to a pro- voked PE or DVT?
  4. What are the 3 things that make up the Vir- chow's triad?
  5. What testing should be done first to look for peripheral artery disease? Anesthesia, immobility, pregnancy, hypercoagulable state, DVT, cancer, hormone replacement Venous stasis Hypercoagulability Endothelial injury ankle-brachial index (BP will be lower on the ankles vs arms)
  6. What test will confirm a diagnosis of PAD? angiography
  7. Symptoms of PAD pale, waxy, hairless legs pain with ambulation that improves with cessation of ambulation
  8. Treatment for PAD stents of bypass of vessels Anti-platelet medications Statins for lipid management Lifestyle modifications and man- agement of co-morbidities (dia- betes)

"I See All Leads" II, III, & eVF: Inferior V1 & V2: Septal V3 & V4: Anterior V5 & V6: Lateral

  1. What procedure is commonly done for A-fib pa- Ablation tients?
  2. When ordering imaging studies, when is con- trast needed?
  3. What are the 5 traits of metabolic syndrome? (Need to have 3 for a diagnosis) When looking for anything vascular
    • Male waist >40in or female waist > 35 in
    • HTN: BP >130/
    • Trigylcerides >
    • Serum HDL < 40 in males or < 50 in females
    • Hyperglycemia: fasting glucose > 100
  4. What is the BP goal for patients with diabetes? 130/80 (to protect the kidneys)
  5. What condition causes a blood sugar > (600-1200), hyperosmolality, and often causes neuro impairment? Hyperglycemia Hyperosmolar State (HHS)
  6. What A1C reading indicates diabetes? 6.5% or greater
  7. When a patient has diabetes, what should their A1C be to be considered "well controlled"?
  1. What should the A1C be before adding a second 8.0% medication?
  1. What is the first-line treatment (medication) for a patient with Type 2 diabetes? Biguanides (Metformin)
  2. Which diabetic medication class is often used as Sulfonylureas (glupizide, gly- a 2nd line treatment but causes hypoglyccemia? buride, glimepiride)
  3. What class of diabetic medication is contra-indi- GLP- 1 cated with a personal or family history of thyroid carcinoma?
  4. Which class of diabetic medications cause glu- cose to be excreted through the bladder and has a side effect of frequent UTIs and yeast infec- tions? SGLT2 inhibitors (end in "gliflozin")
  5. What insulin is long acting? Lantus (20- 24 + hours)
  6. S/S of hypothyroidism fatigue, diflculty losing weight, constipation, cold intolerance, menorrhagia, coarse nails, brittle hair, delayed relaxation phase of DTRs, hypothermia, goiter
  7. What will labs look like for hypothyroidism? Elevated TSH and decreased T
  8. Treatment of hypothyroidism Levothyroxine
  9. Therapeutic/starting doses of levothyroxine Therapeutic is typically 1mcg/kg/day Start low (25-50mcg) and increase as needed
  10. 6 - 8 weeks
  1. Primary and secondary adrenal insufficiency Primary: impairment of the adrenal glands, 80% autoimmune, can be infectious cause or adenoma Secondary: cause by lack of ACTH production in the pituitary gland, abrupt withdrawal of glucocorti- coids
  2. S/S of adrenal insufficiency (Addison's) fatigue, weight loss, hypotensive, hypoglycemia, muscle weakness, nausea, vomiting, abd pain, tan- ning of skin, and salt craving in pri- mary AI *cold, hypotensive, and often stop making urine
  3. Treatment glucocorticoids & mineralocorti- coids (for primary AI) Glucocorticoids only (for sec- ondary AI)
  4. S/S of Cushing's syndrome thin skin, muscle atrophy and weakness, uncontrolled HTN, ab- dominial obesity, striae, rounded face
  5. What is the primary test used to diagnose Cush- 24 hours urine free cortisol ing's syndrome (excess of cortisol)?
  6. diabetes insipidus (DI) Inadequate arginine vasopressin secretion or inadequate renal re- sponse
  1. s/s of diabetes insipidus Excessive thirst and urination
  2. Diagnostic workup for diabetes insipidus 24 - hour testing of:
    • urine osmolality (concentrated urine)
    • creatinine
    • fluid intake (>70mL/kg)
    • urine specific gravity (<1.005 / low)
    • urine glucose (negative) Water deprivation test
    • to ditterentiate between cental and nephogenic Consider MRI of brain
  3. SIADH Overproduction of antidiuretic hor- mone (ADH, aka vasopressin)
  4. SIADH symptoms Impaired water excretion (retain- ing water), - in return water will exceed urine output and cause low sodium (hyponatremia)
  5. Social determinants of health (per health People economic status 2030 - the 5 overarching domains): education access and quality health care access and quality neighborhood and built environ- ment social and community context
  6. Palliative Care Made for comfort - Not end of life care
  • proof that the defendant's breach caused the injury
  1. assault the intentional act of making some fear that you will cause them harm
  2. battery The intentional act of causing phys- ical harm to someone
  3. What is an adverse effect of long-term use of a PPI? osteoporosis and vit B12 deficiency
  4. Barrett's esophagus - Metaplastic columnar epithelium replacing stratified squamous ep- ithelium
  • develops due to chronic GERD
  • increased risk of cancer
  1. Causes and signs of an upper GI bleed Causes: peptic ulcer, esophageal varices, Mallory-Weiss tears S/S: dark, cottee ground emesis or stool
  2. Management of H. Pylori Triple - Quad therapy:
  • 2 antibiotics: clarithromycin, amoxicillin, metronidazole (Flagyl), tetracyclines
  • PPI
  • Sometimes Bismuth is added
  1. Is H. Pylori grain + or Gram - bacteria? Gram -
  2. Gram + bacteria

Staphylococci Streptococci Enterococci

  1. Gram - bacteria - E. coli
- Klebsiella - Proteus - Haemophilus influenzae - Pseudomonas aeruginosa
  1. Microcytic anemia (types) iron deficiency, thalassemias, lead poisoning
  2. What do labs look like with Iron deficiency ane- mia? Low ferritin and increase TIBC
  3. Normocytic anemia (types) Acute blood loss, Anemia of chronic disease
  4. Macrocytic anemia (types) B12 deficiency Folate deficiency Pernicious anemia
  5. Aplastic anemia can present as what type of ane- Normo or macrocytic (get a bone mia?
  6. MCV (mean corpuscular volume), MCH, and RDW marrow biopsy) MCV - size of blood cells MCH - color/redness RDW - red cell distribution - If el- evated, there are immature RBCs. The RDW elevates with almost all anemias expect thalassemia
  7. What meds are used C. Diff? Flagyl or Vanco PO

Surface = right now antigen - active infection/sick

  1. Hepatitis C testing HCV (+) doesn't indicate whether its acute of chronic If viral copies are present, virus is acute
  2. if AST:ALT ratio is 2:1 what may this indicate alcoholic liver damage/alcohol abuse
  3. S/S of diverticulitis - Constant LLQ pain present for sev- eral days
- fever, n/v, (possible constipation or diarrhea) - Rebound tenderness, positive Rovsing's sign, board-like ab- domen
  1. Crohn's Disease vs Ulcerative Colitis Crohn's - mouth to anus UC - only in the colon
  2. Treatment of acute flair of irritable bowel dis- eases steroids
  3. Chronic management of irritable bowel disease 5 - ASA
  4. What is an adverse affect of Zofran? prolonged QT wave
  5. What should an H2 blocker be taken? before meals (spiciest meal)
  6. S/S of macular degeneration Gradual or sudden, painless loss of central vision
  1. S/S of acute angle closure glaucoma painful, injected eye with halos around light May have n/v Elevated eye pressure *True ophthalmologic emergency
  2. S/S of open angle glaucoma Progressive peripheral visual field loss, then central vision loss "Cupping" is noted on fundoscopic exam
  3. S/S of detached retina flashing lights, floaters, shadowy peripheral vision, gray curtain over part of view
  4. What is chronic inflammation of the meibomian Chalazion gland that is painless and described as a "bead" in the eyelid
  5. What is an abscessed hair follicle and/or seba- ceous gland with an acute onset of edematous, erythematous, and warm abscess on the upper or lower eyelid? Hordeolum (stye)
  6. Treatment of chalazion warm compresses (antibiotics are NOT indicated)
  7. treament of hordeolum (Stye) hot/warm compresses 2 - 3 x per day Empiric antibiotics for preseptal cellulitis Possible refer to ophthalmologist for I&D
  1. Mononucleosis often causes adenopathy in which chains? posterior cervical
  2. What reaction is common penicillins are given to Rash someone with Mono?
  3. What can be caused by untreated periodontal abscesses? bacterial endocarditis
  4. First-line treatment of periodontal abscess Augmentin
  5. Retropharyngeal abscess life-threatening infection in the lat- eral pharyngeal space that has the potential to occlude the airway *ENT emergency
  6. Treatment of retropharyngeal abscess IV ceftriaxone or clindamycin
  7. What are the 4 D's of epiglottitis? Dysphagia Dysphonia Drooling Distress
  8. How is epiglotitis treated? Ceftrioxone Oxygen therapy Possible intubation/surgery
  9. Lymph nodes are known as "trash cans" of the body
  10. Lymphoma s/s night sweats, unexplained weight loss, drenching sweats
  11. When treating lymphoma, what is often caused? pleural ettusion or tumor lysis syn- drome
  1. Osteoarthritis is often: unilateral
  2. Rheumatoid arthritis is often: bilateral
  3. How is Rhematoid Arthritis diagnosed? rheumatoid factor (RF)
  4. treatment of rheumatoid arthritis Steroids: early and extremis DMARDs: chronic management
  5. systemic lupus erythematosus s/s butterfly rash, joint pain, fatigue
  6. Treatment of lupus Hydroxychloroquine, NSAIDs, im- munosuppressants, DMARDs, cor- ticosteroids Belimumab Sunlight, diet and exercise, avoid- ance of stress, smoking cessation
  7. Treatment of Multiple Sclerosis (MS) Monoclonal antibodies for chronic managment Steroids for acute flares
  8. myasthnia gravis (MG) Chronic autoimmune disor- der in which antibodies de- stroy nerve/muscle communica- tion, causing chronic long-term muscle weakness
  9. Guillain-barre Acute bacterial or viral infection triggers demyelination ascending vs descending may be fatal if respiratory muscles attected