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Nursing Exam 3 (Peds) Study Guide

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Nursing Exam 3 (Peds) Study
Guide
Cardiac
Meds:
oLasix – I & O, K enriched foods
oDigoxin
Increases contractility, slows heart, dangerous drug with narrow window of safety.
Tale apical pulse X1min
If vomits, do not repeat dose
Dig Toxicity: Anorexia, nausea, vomiting, diarrhea, visual disturbances (Yellow vision<
bradycardia.
Closely monitor for toxicity if on antibiotics
CHF = Causes, S/S, nursing interventions, treatment
oS & S – Tachycardia, diaphoretic, weight gain, cardiac output decreased due to fluid overload
If on CHF, may require O2 during procedures
oCongenital defects: S/S, nursing interventions
oASD – may be asymptomatic but can develop CHF. Oxygenated blood flows back in the right
atrium. Usually with exercise intolerance. Diastolic murmur, risk for atrial dysrhythmias. Can
be treated with a surgical patch closure or cardiac cath can now be performed to dispatch a
closure device.
oCSD – Can cause hypertrophy of the right ventricle from the pressure from the lungs and the left
to right shunting. CHF is common, there is a loud holosystolic murmur.
oCoarctation of the aorta – Narrowing near the insertion of the ductus arteriosus which results
in increased pressure proximal to the defect (head and upper extremities) and decreased
pressure distal to the obstruction (Body and extremities). Patient presents with increased blood
pressure and bounding pulses in the arms, weak or absent femoral pulses and a cool lower
extremities with lower blood pressure.
oA cyanotic = oxygenated blood is not getting out to the systemic circulation, may go into the
lungs.
oTetralogy of Fallot: Four defects are ventricular septal defect, pulmonic stenosis, overriding
aortal and right ventricular hypertrophy. Manifestations are cyanotic at birth. It can start of mid
and progress with time. There is a systolic murmur, moderate in intensity. There can be acute
episodes of cyanosis and hypoxia call blue spells or you may see elevated HR and RR. When
these spells occur you will place the infant in the knee chest position. You will see older
children will automatically go into this position. Treatment is usually done within the first year
of life. Mortality is less then 3 percent. Congestive heart failure is a concern posturer.
Rheumatic fever: Causes, s/s, nursing interventions, treatment.
oInflammatory disease occurs after group A
oB-hemolytic streptococcal pharyngitis
oTreatment of streptococcal tonsillitis / pharyngitis
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Nursing Exam 3 (Peds) Study

Guide

Cardiac

  • Meds: o Lasix – I & O, K enriched foods o Digoxin ▪ Increases contractility, slows heart, dangerous drug with narrow window of safety. ▪ Tale apical pulse X1min ▪ If vomits, do not repeat dose ▪ Dig Toxicity: Anorexia, nausea, vomiting, diarrhea, visual disturbances (Yellow vision< bradycardia.
  • Closely monitor for toxicity if on antibiotics
  • CHF = Causes, S/S, nursing interventions, treatment o S & S – Tachycardia, diaphoretic, weight gain, cardiac output decreased due to fluid overload ▪ If on CHF, may require O2 during procedures o Congenital defects: S/S, nursing interventions o ASD – may be asymptomatic but can develop CHF. Oxygenated blood flows back in the right atrium. Usually with exercise intolerance. Diastolic murmur, risk for atrial dysrhythmias. Can be treated with a surgical patch closure or cardiac cath can now be performed to dispatch a closure device. o CSD – Can cause hypertrophy of the right ventricle from the pressure from the lungs and the left to right shunting. CHF is common, there is a loud holosystolic murmur. o Coarctation of the aorta – Narrowing near the insertion of the ductus arteriosus which results in increased pressure proximal to the defect (head and upper extremities) and decreased pressure distal to the obstruction (Body and extremities). Patient presents with increased blood pressure and bounding pulses in the arms, weak or absent femoral pulses and a cool lower extremities with lower blood pressure. o A cyanotic = oxygenated blood is not getting out to the systemic circulation, may go into the lungs. o Tetralogy of Fallot: Four defects are ventricular septal defect, pulmonic stenosis, overriding aortal and right ventricular hypertrophy. Manifestations are cyanotic at birth. It can start of mid and progress with time. There is a systolic murmur, moderate in intensity. There can be acute episodes of cyanosis and hypoxia call blue spells or you may see elevated HR and RR. When these spells occur you will place the infant in the knee chest position. You will see older children will automatically go into this position. Treatment is usually done within the first year of life. Mortality is less then 3 percent. Congestive heart failure is a concern posturer.
  • Rheumatic fever: Causes, s/s, nursing interventions, treatment. o Inflammatory disease occurs after group A o B-hemolytic streptococcal pharyngitis o Treatment of streptococcal tonsillitis / pharyngitis

▪ Antibitoics ▪ Anti-inflammatories ▪ Bedrest.

  • Kawasaki Disease: o Etiology is unknown, though nor known to spread from person to person it is seen in geographic and seasonal outbreaks. o Mucocutaneous lymph node syndrome. o Acute systemic vasculitis of unknown cause o %75 of cases are children under age 5 o Self-limiting o Increased risk of coronary artery aneurysm o Acute Phase – Abrupt high fever not responsive to antibiotics, irritability, rash, red eyes, peeling hands and feet, irritability, red tongue. o Subacute phase – Resolution of fever and lasts until all clinical signs of KD have disappeared. This phase is where the child is at greatest risk for developing coronary artery aneurysms. o Convalescent phase – All signs and symptoms have resolved but lab values remind abnormal until around 6-8 weeks later. o Treatment ▪ Globulins ▪ Aspirin o Nursing interventions ▪ Light weight clothing ▪ Monitor temperature ▪ Nutrition: Bland foods, avoid citrus or other irritating foods.
  • Cardiac Procedures ▪ Nursing interventions ▪ Postprocedural care – check pulses especially where?? (Below the catherization site) Check Temp and color of extremity – could indicate arterial obstruction. ▪ Vital signs - Pulse for one full minute – Check for Dysrhythmia and bradycardia. - Blood pressure can indicate hypotension which could mean hemorrhage. - Children are able to maintain blood pressure longer then adults. - Dressing check – For Bleeding or hematoma - Fluid intake – Dehydration due to NPO and diuretic action of the dyes Respiratory
  • Growth and development issues o How does ages affect anatomy and predispose to respiratory problems?
  • Nasopharyngitis: “common cold” o Caused by numerous viruses o RSV, rhinovirus, adenovirus, influenza and parainfluenza viruses o Fever: varies with age of the child o Home management: varies with age

o o Nursing care management ▪ Relieve the pain (AOM) ▪ Facilitate drainage ▪ Educate the family ▪ Provide emotional support o Prevention of recurrence ▪ Take all antibiotics ▪ Hold child upright when feeding ▪ Do not prop bottle o

  • Causes, S/S, nursing interventions, education, and treatment. o Epiglottis ▪ Signs – Leaning forward (Tripod position), drooling ▪ SOB with shortness with restricted breath sounds is an ominous sign / imminent respiratory arrest ▪ Treatment - Allergen control - Drug Therapy - Chest physiotherapy
  • RVS – Respiratory Syncytial Virus o Pathophysiology ▪ The bronchiole mucosa swell and lumina are filled with mucus and exudate o Clinical manifestations ▪ URI-rhinorhea and low-grade fever ▪ Cough-non productive, prosysmal ▪ Apnea o Diagnostic evaluation ▪ ELISA-enzyme-linked immunosorbent assay o Therapeutic management ▪ Symptomatically ▪ Keep nares clear ▪ Oxygen mist ▪ IV fluids o Prevention of RSV, prophylaxis
    • Nursing Care with RVS o Contact and standard precautions

o Educate Family o Monitor oxygenation o Bronchodilator therapy o IV therapy

  • Pertussis (Whooping Cough) o Caused by Bordetella pertussis o In United States it occurs most often in children who have not been immunized o Highest incidence in spring and summer o Highly contagious o Risk to young infants o Vaccines
  • Asthma o Chronic inflammatory disorder of airways o Bronchial hyperresponsiveness o Episodic o Limited airflow or obstruction that reverses spontaneously or with treatment o Etiology and pathophysiology o Asthma Severity Classification in children 5 or older ▪ Step I: mild, intermittent asthma ▪ Step II: mild, persistent asthma ▪ Step III: moderate, persistent asthma ▪ Step IV: severe, persistent asthma o Diagnostic evaluation ▪ Pulmonary function tests ▪ Skin testing for allergens o Therapeutic management ▪ Allergen control ▪ Drug therapy o Nursing care management ▪ Avoid allergen ▪ Relieve bronchospasms ▪ Provide acute asthma care o Drug Therapy for Asthma ▪ Long-term control medications ▪ Quick-relief medications ▪ Metered-dose inhaler ▪ Corticosteroids ▪ Cromolyn sodium ▪ Albuterol, metaproterenol, terbutaline ▪ Long-term bronchodilators (Servent) ▪ Theophylline: monitor serum levels ▪ Leukotriene modifiers o Asthma Interventions
  • Present in almost all CF patients, but onset/extent is variable
  • Stagnation of mucus and bacterial colonization result in destruction of lung tissue
  • Tenacious secretions are difficult to expectorate—obstruct bronchi/bronchioles
  • Decreased O 2 /CO 2 exchange
  • Results in hypoxia, hypercapnea, acidosis
  • Compression of pulmonary blood vessels and progressive lung dysfunction lead to pulmonary hypertension, cor pulmonale, respiratory failure, and death ▪ Infectious Pathogens _- Pseudomonas aeruginosa
  • Burkholderia cepacia
  • Staphylococcus aureus
  • Haemophilus influenzae
  • Escherichia coli
  • Klebsiella pneumoniae_ ▪ Respiratory Progression
  • Gradual progression follows chronic infection
  • Bronchial epithelium is destroyed
  • Infection spreads to peribronchial tissues, weakening bronchial walls
  • Peribronchial fibrosis
  • Decreased O 2 /CO 2 exchange ▪ Further Respiratory Progression
  • Chronic hypoxemia causes contraction/hypertrophy of muscle fibers in pulmonary arteries/arterioles
  • Pulmonary hypertension
  • Cor pulmonale
  • Pneumothorax
  • Hemoptysis ▪ Gastrointestinal GI tract effects
  • Thick secretions block ducts, cystic dilation, degeneration, diffuse fibrosis
  • Prevents pancreatic enzymes from reaching duodenum
  • Impaired digestion/absorption of fat, steatorrhea
  • Impaired digestion/absorption of protein, azotorrhea
  • Endocrine function of pancreas initially stays unchanged
  • Eventually pancreatic fibrosis occurs;
  • may result in diabetes mellitus
  • Focal biliary obstruction results in multilobular biliary cirrhosis
  • Impaired salivation ▪ Clinical Manifestations of GI tract
  • Pancreatic enzyme deficiency
  • Progressive chronic obstructive pulmonary disease associated with infection
  • Sweat gland dysfunction
  • Failure to thrive
  • Increased weight loss despite increased appetite
  • Gradual respiratory deterioration ▪ Presentation
  • Wheezing respiration, dry nonproductive cough
  • Generalized obstructive emphysema
  • Patchy atelectasis
  • Cyanosis
  • Clubbing of fingers and toes
  • Repeated bronchitis and pneumonia
  • Meconium ileus
  • Distal intestinal obstruction syndrome
  • Excretion of undigested food in stool; increased bulk, frothy, and foul
  • Wasting of tissues
  • Prolapse of the rectum
  • Delayed puberty in females
  • Sterility in males
  • Parents report children taste “salty”
  • Dehydration
  • Hyponatremic/hypochloremic alkalosis
  • Hypoalbuminemia ▪ Diagnostic Evaluation
  • Quantitative sweat chloride test
  • Chest x-ray
  • Pulmonary function test
  • Stool fat and/or enzyme analysis
  • Barium enema ▪ Goals
  • Prevent/minimize pulmonary complications
  • Adequate nutrition for growth
  • Assist in adapting to chronic illness ▪ Respiratory Management
  • Bronchodilator medication
  • Forced expiration
  • Aggressive treatment of pulmonary infections
  • Home intravenous antibiotic therapy
  • Aerosolized antibiotics
  • Hemoptysis
  • Nasal polyps
  • Steroid use/nonsteroidal antiinflammatory
  • Transplantation ▪ GI MANAGEMENT
  • Replacement of pancreatic enzymes
  • High-protein, high-calorie diet as much as 150% recommended dietary allowance
  • Intestinal obstruction
  • Reduction of rectal prolapse
  • Salt supplementation ▪ Prognosis of CF (Cystic Fibrosis)
  • Estimated life expectancy for child born with CF in 2005 is 36.5 years
  • Maximize health potential
  • Nutrition
  • Prevention/early aggressive treatment of infection
  • Pulmonary hygiene
  • New research: hope for the future

▪ When child’s weight, age or condition is deemed appropriate, surgeons close the stoma, remove the diseased portion of the bowel, and perform a pull-through procedure, which repairs the colon by connecting functional bowel to the anus. Usually establishes fairly normal bowel function. ▪ Overall, prognosis is very good ▪ Most infants with Hirschsprug’s disease achieve good bowel control after surgery, but small percentage of children may have lingering problems with soilage or constipation. These infants are also at higher risk for an overgrowth of bacteria in the intestines, including subsequent episodes of enterocolitis, and should be closely followed by a physician. o Nursing considerations for Hirschsprung’s Disease ▪ Foster infant parent bonding ▪ Prepare parents for medical-surgical procedure ▪ Assist them with ostomy care they will perform at home, if needed ▪ Measure abdominal circumference at the umbilicus ▪ Teach preschool and older child about ostomy using concrete terms and visual age- stress temporary ostomy ▪ Post-operative care-stoma care, Foley catheter ▪ Teaching concerning ostomy and return for additional surgery ▪ Before surgery child may need to be built up with low fiber, high calorie, high protein, diet and possibly TPN ▪ Newborn bowel is sterile no prep necessary ▪ Older children need repeated saline enemas and decreasing bacterial flora with systemic antibiotics and colonic irrigations using antibiotic solutions and oral antibiotics for the prep for stage 2 ▪ Frequent monitoring of VS due to possibility of enterocolitis-fever, increasing abdominal distention vomiting, increased abdominal tenderness, shock irritability could mean bowel is perforated ▪ Progressive distention of the abdomen is a serious sign, that is why the nurse needs to measure the abd cir. And then measure every time take vitals, leave tape measure under child mark spot where measurement for realiablity ▪

  • Roto virus o Cause ▪ Most diarrhea is spreat by the fecal-oral route o Treatment ▪ Teach personal hygiene ▪ Clean water supply / protect from contamination ▪ Careful food preparation ▪ Handwashing
  • GER o Defined as transfer of gastric contents into the esophagus

o Occurs in everyone o Frequency and persistency may make it abnormal o May occur without GERD o GERD may occur without regurgitation o S/S ▪ Spitting up, regurgitation, vomiting, which can be forceful ▪ Excessive crying, irritability, arching of back, stiffening ▪ Weight loss, failure to thrive ▪ Cough, wheeze, gagging, choking with feedings) ▪ Can occur throughout the day, but most frequently after meals and at night. What is occurring is that the lower esophageal sphincter (LES) and be affected by pressures that can vary due to gastric distention, increased abdominal pressure caused by coughing, CNS disease, delayed gastric emptying, hiatal hernia and gastrostomy placement (Transient relaxation) o Treatment ▪ Small, frequent feedings ▪ Sitting upright ▪ Thickening the formula. The consistency for thickening would be 1 tsp to 1 tablespoon of rice cereal per ounce of formula. ▪ Children older than 1 year benefit to left side position during sleep and the elevation of the head of the bed. Also keeping child upright for 1 hour after feedings help. o Teaching ▪ Administer Protonics 30 minutes prior to feedings, make sure parents understand that it is not given before each feeding. ▪ Majority of infants have improvement by age 12 to 18 months of age and require only conservative lifestyle changes of medical interventions. ▪ Older children – Avoid caffeine, chocolate and spicy foods, citrus and peppermint ▪ Tobacco and alcohol may aggravate condition

  • Cleft Lip & Palate repair o Multifactoral inheritance: genetics & environment o They are common to all populations: o CL/P most common in Native Americans o CL/P least common in African Americans o Maternal smoking in first trimester believed to be cause in 11% to 12% of all cases o Also physical & emotional stress; tobacco use; teratogenic drugs & nutritional deficits o Failure of the maxillary and premaxillary process to come together between the third and twelfth week of embryonic development. CL and CP are distinct malformations that occur at different times during developmental process. o Clefts can be unilateral or bilateral o CL may vary from a small notch to a complete cleft extending into base of the nose o Deformed dental structures associated w/CL o CP alone occurs in midline and may involve the soft and hard palates o CP with CL may involve the midline and extend into the soft palate on one or both sides

o Proper positioning – Arms restrained at elbows (NO NOS) o Adequate analgesia o Clear liquids initially: progress to full feeding (May need to use other method for feeding besides nipple since nipple could breakdown sutures) o Cleanse suture site with cotton tipped swap in saline o Thin layer of antibiotic ointment

  • Cleft Palate o Surgical Correction & Prognosis of CP ▪ Post-op: allow to lie on abdomen ▪ Resume feedings by bottle, breast, or cup shortly after surgery ▪ Avoid suction or other objects in mouth ▪ Elbow restraint (4-6 weeks) ▪ Assess and medicate for pain ▪ Discharge on blenderized or soft diet ▪ Caution against hard items: toast, hard cookies, chips ▪ With the cleft palate repair, surgery is usually postponed until 12 months to take advantage of palatal changes that take place with normal growth; and before the child develops faulty speech habits. ▪ child to lie on abdomen to prevent aspiration. ▪ To preserve the integrity of the surgical site we aviod suctioning of the mouth or placing other objects in the mouth such as straws and thermometers that may cause trauma. The child will go home with elbow restraints for 4 to 6 weeks, so teaching parents to release the restraints one at a time and monitor for skin break down. Teach parents to avoid allowing the child to have any hard items in his or her mouth such as toast, hard candy, etc. that may cause trauma to the surgical site. ▪ o Resume feedings by bottle, breast, or cup shortly after surgery o Avoid suction or other objects in the mouth o Elbow restraint (4-6 weeks) o Assess and medicate for pain o Discharge on blender or soft diet o Caution against hard items, toast, hard cookies, chips o Nursing considerations CL / CP ▪ Promote optimal nutrition ▪ Psychological & Intellectual Development ▪ Aesthetics ▪ Hearing & Speech ▪ Tooth Development ▪ Immediate care of URI’s ▪ POST SURGICAL: - No trauma; minimal to no pain - Optimum nutrition - No complications - Family support - At home care
  • Pyloric stenosis o Constriction of pyloric sphincter with obstruction of gastric outlet o Pathophysiology o Thickening of the circular muscle of the pylorus causing constriction of the pylorus and obstruction of the gastric outlet o Usually develops in first few weeks of life o Etiology unknown however genetic predisposition o More common in firstborn, full-term, Caucasian males o Obstructive disorder, thickening of the circular muscle of the pylorus causing constriction of the pylorus and obstruction of the gastric outlet. You will start to see signs and symptoms develop in these infants between 2 and 5 weeks of life. o S/S ▪ Projectile vomiting up to 30 minutes after eating ▪ Palpable olive like mass ▪ Visible peristatic waves ▪ Metabolic alkalosis. o Treatment ▪ Infant must undergo surgical treatment to correct this obstruction ▪ Preoperatively it is important to make sure that is there is dehydration and metabolic alkalosis occurring it has to be corrected before surgery. ▪ Postoperatively, feedings will begin about 4-6 hours after. Will need to be small, frequent feedings with glucose water and electrolytes.
  • Intussusception o Occurs when one portion of the bowel slides into the next, like pieces in a telescope. Creates obstruction in the bowel, with the walls of the intestines pressing against one another. Leads to swelling, inflammation and decreased blood flow to the intestines involved. o S/S ▪ Severe colicky abdominal pain in a child with vomiting and currant jelly like stools (Mucus and blood) o Treatment ▪ Radiologist guided pneumo-enema ▪ Ultrasound guided saline enema ▪ Watch for normal bowel movement after procedure.
  • TEF ( Esophageal Atresia and Trachoesophageal Fistula ) o Failure of esophagus to develop as continuous passage o May occur separately of in combination o Etiology and pathophysiology unknown

usually noted on inspection and palpation of the abdomen. Usually closes spontaneously by 1-2 years of age. ▪ As nurses, discourage parents from using home remedies (belly bands or coins) ▪ Inguinal= protrusion of abdominal contents through the inguinal canal into the scrotum ▪ Detected as painless inguinal swelling of variable size ▪ Surgical closure of inguinal canal o

  • Appendicitis o S/S, fever, vomiting, abdominal pain and increased WBC. Ultrasound will show the presence of fluid around the appendix. On CT there will be presence of an enlarged appendiceal diameter. o First symptoms that will be apparent: Periumbilical pain, nausea, RLQ pain, later vomiting with a fever. o Assess severity of pain – change in behavior – refuse to play. o Younger Child – Rigid position, side-lying, knees flexed at abdomen, decreased ROM of right Hip o Older child can point to spot of pain. o NO LAXATIVES, ENEMAS OR HEAT! = Stimulates bowel motility and increases risk of perforation o Post Op = IV Fluids, NPO, NG until intestinal activity returns, check bowel sounds, antibiotics, if ruptured hospitalized 7-10 days.
  • Cardiac Disease o Four characteristics ▪ Steatorrhea ▪ General Malnutrition ▪ Abdominal distention ▪ Secondary vitamin deficiencies o TREATMENT = Gluten free Diet (Known as low gluten) ▪ Eliminate wheat, rye, barley and oats ▪ Substitute corn and rice. ▪ S/s of dehydration – nursing interventions and education
  • Ingestion of poisons o Usually under 6 years of age o Medications o Cosmetics o Cleaning products o Plants o Foreign bodies o Parents should have poison control telephone number posted. o Glomerulonephritis ▪ Most common caused by strep ▪ Oliguria – Very small amount of urine

▪ Edema ▪ Hypertension and circulatory congestion ▪ Hematuria ▪ Bleeding in upper urinary tract – smoke urine or tea colored ▪ Proteinuria ▪ Increased amount of protein = increased severity of renal disease ▪ Acute Glomerulonephritis AGN ▪ Acute glomerulonephritis due to Streptococcal

  • Most common of the postinfectious renal diseases in childhood
  • Most common 6-7year olds, males
  • There is 10 to 21 days between streptococcal infection and clinical manifestations.
  • Infection may originally manifest in different region of body (pharynx or skin- impetigo).
  • Glomerular deposition=Antigen-antibodies lodge in glomeruli due to immune reaction causing inflammation impairment of the kidney to filter urine. Cause still questionable-immune complexes are deposited in glomerular basement membrane after recent strep infection
  • Glomeruli become edematous and infiltrated with leukocytes, occlude capillaries
  • Decrease in urine filtration-water accumulates and retention of sodium leading to circulatory congestion, hypertension and edema, primarily periorbital and peripheral ▪ Therapeutic Management
  • Early recognition and treatment
  • Children with normal BP and urine output can be treated at home
  • Moderate sodium restriction and fluid restriction-depending on stage of disease
  • Potassium restricted when oliguric
  • VS-to assess for early complications
  • Body weight-fluid balance
  • I&O measured
  • Acute hypertension-BP every 4-6hrs
  • Antibiotics only with persistent strep
  • Rest
  • Volume and character of urine ▪ Prognosis
  • Almost all children diagnosed with acute poststreptococcal glomerulonephritis recover completely
  • Specific immunity is conferred
  • Subsequent recurrences are uncommon
  • Some children have been reported to develop chronic disease ▪ Nursing Care Management
  • Manage edema
  • Daily weights
  • Accurate input and output
  • Daily abdominal girth
  • Nutrition
  • No added salt if hypertensive
  • Limited activity
  • Anti-hypertensives as needed
  • This reduces the serum albumin level, decreasing the collodial osomotic pressure in the capillaries. The vascular hydrostatic pressure exceeds the pull of the colloidal osomtic causing fluid to accumulate in the interstitial spaces (edema) and body cavities, patricularly the abd cavitiy (asicitis).
  • The vascular fluid decreases and this stimulates the renin-angiotensin system- this then secretes antidiuretic hormone and aldosterone. This increases the absorption of NA and Water.
  • There is also an unexplained increase in serum lipids. ▪ Nursing considerations for Nephrotic Syndrome
  • Monitor fluid retention or excretion
  • Strict I and O
  • Examine urine for albumin
  • Daily weight
  • Measurement of Abdominal girth
  • Assessment of edema-eyes, dependent areas, degree of pitting, color and texture of skin
  • Vital signs-shock or infection ▪ Family issues wit Nephrotic Syndrome
  • Chronic condition with relapses
  • Developmental milestones
  • Social isolation
  • Lack of energy
  • Immunosuppression/protection
  • Change in appearance caused by edema
  • Self-image ▪ o Enuresis – most common is bed wetting ▪ Inappropriate voiding 2 times a week for 2 months ▪ Enuresis is the intentional or involuntary passage of urine into bed at night or clothes during the day in children who are beyond the age when voluntary bladder control should normally have been acquired. ▪ Occurs twice a week for three months and child must be at least 5 years old ▪ More common in boys ▪ Structural causes, diabetes, sickle cell disease, ▪ Strong familial tendency ▪ Imipramine-tricylic antidepressant ▪ ▪ Primary/ secondary/ diurnal/ nocturnal ▪ Identify potential causes ▪ Fluid restrictions, bladder training, enuresis alarms ▪ Imipramine ▪ Emotional support ▪ o Hypospadias / epispadias ▪ DO not circumcise ▪ Hypospadias- uretharal opening located behind glans penis or anywhere along ventral surface of penile shaft.

▪ Surgery is to produce a sexually adequate organ, improve physical appearance and allow child to void standing up.l ▪ Epispadias- Meatal opening is located on dorsal surface of the penis. Surgical correction usually includes penile and urethral lengtening and bladder neck reconstruction. o UTI = Treatment – cotton panties, no bubble baths, increase fluids.

  • Musculoskeletal
  • Always Check the 5 P’s
  • Immobilization causes venous status
  • Clubfoot – Treatment is serial casting changed every one to two weeks and will require treatment for life (Not casting for life)
  • Juvenile idiopathic arthritis o Use immunosuppressant
  • Congenital hip dysplasia o Pavlik harness is used which the child wears 23 hours a day. Parents should be taught to massage skin once a day.
  • Duchenne’s muscular dystrophy o Genetic so parents should be tested o Usually die by school age **- Growth and Development
  • Factors influencing Development** o Genetics o Nurition o Prenatal & Environmental Factors o Family & community o Cultural - Anticipatory o Process of understanding upcoming developmental needs & teaching caregivers about those needs o Includes ▪ Health habits ▪ Prevention of injury & illness ▪ Prevention of poisonings