Telemetry-Capstone Nursing Study Guide, Exams of Nursing

Telemetry-Capstone Nursing Study Guide

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Telemetry-Capstone Nursing Study Guide
Normal Sinus Rhythm
Heart Rate: 60-100 bpm
Regularity: Regular
PRI: .12-.20 seconds
QRS: <.12 seconds
Normal Sinus Bradycardia
Heart Rate: <60 bpm
Regularity: Regular
PRI: .12-.20 seconds
QRS: <.12 seconds
Normal Sinus Tachycardia
Heart Rate: >100 bpm
Regularity: Regular
PRI: .12-.20 seconds
QRS: <.12 seconds
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Telemetry-Capstone Nursing Study Guide

Normal Sinus Rhythm Heart Rate: 60-100 bpm Regularity: Regular PRI: .12-.20 seconds QRS: <.12 seconds Normal Sinus Bradycardia Heart Rate: <60 bpm Regularity: Regular PRI: .12-.20 seconds QRS: <.12 seconds Normal Sinus Tachycardia Heart Rate: >100 bpm Regularity: Regular PRI: .12-.20 seconds QRS: <.12 seconds

Normal Sinus Arrhythmia Heart Rate: 60-100 bpm; can be < Regularity: Irregular PRI: .12-.20 seconds QRS: <.12 seconds Premature Atrial Contraction (PAC) Heart Rate: Depends on underlying rhythm Regularity: Interrupts the regularity of underlying rhythm P-Wave: can be flattened, notched, or unusual. May be hidden within the T wave PRI: measures between .12-.20 seconds and can be prolonged; can be different from other complexes QRS: <.12 seconds Atrial Tachycardia (SVT) Regularity: R-R intervals are constant; Regular Rate: artial/ventricular rates are equal; heart rate is between 150- bpm. P-Wave: One P Wave in front of every QRS; may be flattened or notched; because of the rapid rate, the P waves can be hidden within the T waves PRI: .12-.20 seconds and constant QRS: <.12 seconds

PRI: Since no P wave is present, PRI is not determined QRS: <.12 seconds Junctional Rhythms -Occurs when the AV node takes over as the primary pacemaker in the heart rather than the SA node. AV node takes over when is moves faster than SA node. Rate: 40-60 bpm; Accelerated Junctional: 60-100 bpm; Junctional Tachycardia: 100 bpm or greater P Wave: If before QRS, P wave will be inverted. P Wave can also be hidden within the QRS complex. P Wave is usually <.12 seconds QRS: <.12 seconds What are the four Supra-Ventricular Tachycardias (SVT)? Sinus Tachycardia (100-160 bpm) Atrial Tachycardia (150-250 bpm) Atrial Flutter (150-250 bpm) Junctional Tachycardia (100-180 bpm) First Degree Heart Block Regularity: depend on the rhythm Rate: Depend on underlying rhythm P Waves: Upright and Uniform; each P Wave will be followed by a QRS complex

PRI: constant across entire strip, but always > .20 seconds. QRS: < .12 seconds Second Degree Heart Block (Wenckebach) Regularity: R-R Wave is irregular; R-R interval gets progressively shorter as PRI gets progressively longer Rate: Ventricular rate is slightly slower than normal; atrial rate is normal P-Waves: upright and uniform; some p waves are not followed by the QRS complex PRI: gets progressively longer until one p wave is not followed by a QRS complex; after the blocked beat, cycle starts over QRS: < .12 seconds Second Degree Heart Block (Morbitz) Regularity: if conduction ratio is consistent, R-R interval will be constant and rhythm, regular. If conduction ratio varies, the R-R will be irregular Rate: atrial rate is usually normal; ventricular rate will be in bradycardia P Waves: upright and uniform; always be more P waves than QRS PRI: constant; might be longer than normal QRS: <.12 seconds

Regularity: chaotic Rate: cannot be determined P Waves: no P waves present PRI: no PRI QRS: no discernible QRS complexes Asystole No electrical activity; only a straight line 3rd Degree Heart Block Regularity: Regular Rate: 40-60 bpm if junctional; 20-40 bpm if focus is ventricular. P Wave: upright and uniform; more p waves than QRS complexes PRI: no relationship between p waves and QRS complexes QRS: < .12 seconds if junctional; > .12 seconds if ventricular Bundle Branch Block (Left) Wide QRS (>.12 seconds) Left Bundle Branch ("M") Can deteriorate to a 3rd Degree HB

Bundle Branch Block (Right) Wide QRS (>.12 seconds) Right Bundle Branch Block ("V") Can deteriorate to a 3rd Degree HB Lead Placement Left: Smoke (Black) over Fire (Red) Right: Snow (White) over Grass (Green) Center: Chocolate (place a little off center for possible CPR) Sinus Tachycardia Etiology/Clinical Signs Etiology: -Physiologic demand for oxygen -Sympathomimetric Drugs -Fever -Pain Clinical Signs: -increased HR; increased oxygen demand Sinus Tachycardia Treatment -May resolve with treatment of underlying cause -Digoxin, Beta Blockers (-olol), Verapamil -Vagal Maneuver

We have an expert-written solution to this problem! Atrial Flutter Etiology/ Clinical Signs Etiology: -occurs w/ heart disease -CAD -Valve Disorders Clinical Signs: -may cause thrombus -"saw tooth" -250-400 bpm Atrial Flutter Treatment -Give anticoagulants (faster the HR, more risk for thrombus) -treat underlying cause -digoxin (slows rate by enhancing AV block) -Quinidine (supresses atrial ectopic block) -Amiodarone -Calcium Channel Blockers (Cardizem)/Beta Blockers (-olol) -consider cardioversion Atrial Fibrillation Etiology/Causes Etiology: -Advanced Age -Valve Disorders -cardiomyopathy Causes: -chocolate (theobromine-stimulant) -sleep apnea -athletes -tall athletes

-aging heart -men more than women Atrial Fibrillation Treatment

  1. Amiodarone
  2. Calcium Channel Blockers, Beta Blockers, digoxin
  3. Synchronized cardioversion if unstable
  4. radio frequency catheter ablation
  5. anti-coagulation therapy
  6. Cardizem Amiodarone May cause liver, lung damage, and worsening of arrhythmias. Pt to report SOB, wheezing, jaundice, palpitations, lightheadedness Rhythms for cardioversion
  7. A-Fib
  8. A-Flutter
  9. SVT Electrical Cardioversion Tx of choice if pt has a hemodynamically unstable tachydysrhythmia; unstable ventricular tachycardia w/ a pulse; prevention of life- threatening dysrhythmias; cardioversion can be planned or emergent; proper cardioversion will correct pt dysrhythmia w/ minimal discomfort and maximum safety Post Cardioversion Care Same as when a pt is in A-Fib If elective, digoxin is usually withheld for 48hrs prior to cardioversion to prevent dysrhythmias after procedure
  1. VT w/ a pulse: cardiovert
  2. monitor more closely
  3. prepare cardioversion (oxygen, lidocaine, treat cause)
  4. VT w/o a pulse: defibrillate (call code) Torsades De Pointes Treatment IV Magnesium Ventricular Fib (Etiology, Clinical Signs)
  5. Same as VT, PVC
  6. Surgical Manipulation of heart
  7. Failed cardioversion
  8. Same as cardiac arrest
  9. EKG is disorganized rhythm Ventricular Fib Treatment
  10. IMMEDIATE DEFIBRILLATION X
  11. CPR
  12. SURVIVAL IS <10% FOR EVERY MINUTE THE PT REMAINS IN V-FIB SCREAM (acronym) for VFib and VTach
  13. Shock Q2min
  14. CPR after shock (compressions followed by resp 30:2) for 2min
  15. Rhythm check after 2 min of CPR and shock again if indicated
  16. Epinephrine or vasopressin
  17. Antiarrythmic medications: Amiodarone/Lidocaine
  18. Magnesium Sulfate

Cardiac Arrest Ventricular Asystole due to VFib Etiology: trauma, overdose, MI Clinical Signs: asystole or VFib, no definable waves, absence of VS Ventricular Asystole TEA: trans-cutaneous pacemaker, epinephrine, atropine 1st Degree Heart Block Causes May be normal variant; inferior wall MI; drugs: verapamil or digoxin 1st Degree Heart Block Treatment Monitor; Observe for symptoms 2nd Degree Heart Block Causes organic heart disease, MI, Dig Toxicity, Beta and Calcium Blockers 2nd Degree Heart Block Treatment Monitor HR, Atropine, Temp Pacemaker, Avoid meds that decrease conductivity 3rd Degree Heart Block Causes Organic Heart Disease, MI, Drugs, Electrolyte Imbalance, Excess Vagal Tone 3rd Degree Heart Block Signs & Symptoms Extreme Dizziness, Hypotension, Syncope, Decrease CO, Altered Mental Status 3rd Degree Heart Block Treatment Pacemaker (temporary or permanent)