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thsi | eh | og ‘waa Ih nina ee > a vessels ere + Peipheral Uiswe_restilance _Cextornal work ) a G pumptng of heart. __ 4 Chambers 2 Aria (upp er) FA gue. 2 vorniuer ( Te 0 “Pofoventai tar valves md Aafcusp' rd Cangnt) tad | Blurspi'd. (left? cMAral valve + / deny a 3... Jy | ; > | ie y) R> iv ; 5 | | 2 =. fu\limona 25/$ mm Hunk ‘' ung 7 + [Layers at _ | ae 4 7 é 3) one a TWheRies, OF Cone ruses = >> Syncopiuey y _Funaiions of heart °- ulrinclet Jeinee Muscle pun ms of inkevcalat — Paterieas 5) dtaibuting Channels vi ae ae Capiltarles eae 0 on whith eneeE Ty 000 “yoy Sting, | ds Conductivity ca lata, NA) potenrial C_Réspon 2. | éxtitabiyay Oi 195 ps 3. Rutormytomtcity a BY. iontartfitty > =D long yetoourve pond mt 7 Sep & fenaional synvy umn SP Oinietat, depolarisation | a: ‘Dh vensforuby.9 (erat Mises (2) tnvtfal repolast ator = 8 extrasystole and Bt =a Plateau (4a ~9 jompensatory pause SS] Ofinar pepovasatir | q. AU OR Rofl waw = sity 4oes — ] io. | Stor vse e Freee tin i He | Summation 2. | Gffeck of preload ~lGffece oF APeerload: 3 __# fmportomie F long Refractive postock sida 4 east wlll never. i” fatigue beaut 't i novi me for next tontgachon pes “ult cnporlg pose Fe erate reo — FAR None a G ff tsue respons, Nia segpends to fs maximum oF ft does not _sespond at all. ‘The magnitude — of response dd tisue to stil seroatins d ) ee PE sive <4 Fyyespeuttve oF claength of stu? oid tohpufl 8 | & Ve oo Ee a ae bisa 7 p } Vi iS fs f} Oy Maly MULE eT ae presure Changes | | | why | card Cpu ea Soke volume - amb. of bod gjeuied by venmines fo ® one peat ? ot #“ _[eavaiat QUEPUt © Stoke volume X heast rate _| i ma oy Foxye ans dyminney N per Qo- 15m “faa? ms : + Casdlay indey amt- oF blood pumped ouk by ventrities per min. per mt- %. of body Surface. __¥ fauors affecting Cardin seBpubrin ail ¢ Phyo! gad v « Parhogoooad ane 1 hye 2. Gender 3. Diusnal variatieq 4 Enuisonmental temp 5. emotions) CS ff Yours Afuctiry Rah fr {wpe plyrioteyical Lateral pesuse, “exerted: by th the bloocl + putroogtca HUE, on rae. veseh was | | BR: Sv Xr HR Sua eee 3c tye) = 1) Systoue. os work done (110-140) ng in) Diao ~_ penipnene| heart desi{arnee ae 0=% eT) Fulse pressure = Stroke volume ake MMC WY) man bli —~_peF fasion pressure Lies Hottc prsgune = ERT diadiau +) puve Ms Je in : Aycan + Sou team Cm (663) Cen Vide J Hood PIAL antesmudiate. K@ nen. wom tev 44) meth. —in hited ¥elsels— 4 SHORT TRH I) Basoverepov Reflex 1 [Fema pease Yo ee ™ cayohd sinw Y AOE Sfnuy both Stattdnes newes a" 4 0 canal neaver d vasocu ltton by vasomotor centye (VME) sre 7 Biogd presure fF LD CECE EE | Aaa nh Te eeeeg pee: oKUS (ovules 0), chemoeet tows elex ft WV. gee | ob pte Oypoxia? (BP vb Dai Se ee {oi | ] nyo ( hyper capris) rn (2 a Hf Mj ae Tye (due fo lactt dud Yppuvate | | (data beclies im ; 2) Pootie bodies ‘i ED i. : nesves : qin g 10 nerve . ya anne. VMC (Abdali vasounsniinoy. 4 BPTT o2fa5/ey | ie f solar préssull - reLep lors - 7 sititn veep tors. 1M bot (3) _(atdiopulionnary [nartal shewh fete the Arita 4 the pulriopayy OYVEYT Es. 2 Milanese arterial préssure reLeptors : absla_ Ob urd. ahh ™m AS his +0 : eee + 2 — Volume. atrial styetun SeLePrOTS . 6 Gicit veflencs paraliel + pam a 7. eae sae SE P ata tee a Va RE ss volume ~—~—~-«dUt) Bainbyi oe ina WY Bainbridge reflex ; (peer ae __ tthestases heavt ate | BP low ae sen a blood rvs for pardl. ay ADH WY eat ag gir, the ne ae TERM MECHANISM, ees Renin . L fponanegen 1. Renal Body tlurdl | pessure Control mechani _\Y Pyessuve dluvesis Reml Body flusal presuve cntvo | a a Pressure diwresrs / Nabvivmat diuresis [\ TBP A WERT? 4 Unive T+ BPLY = \_ ofltst. { bay Renin. Angidensin menanism . _ Shormone decreased BP | s) Kidney ay Renin (From kidney ) a lotens| tho en L (Renin substyate) Gryrotersin I ie Ss converting —* 7 - Angi Dkensin Tr eae i a Cs vasoconstrichay ne ttivated } | tropverts Pty OE Tiss “ Reltntton ee angtevensin Tig uA ae ee BLE 0 High pressure th the vessels - vessel become streihed. = 2 fires Reverse sires (ppt) _ ec value tong ad\usbynent - relaxation | dilation of blood vessels - Fall in BP wjeeabiel aetna = as G (ow pressure - 1s Stress on the vessel voall - vasvalar fa AS 0 tone adjustment - tiqvivening of blood vessel waits ( Can ‘Abdominal _Comptesion veFlex G when a barol ceptor OR chemoreceptor reflex 15 elit |__ nerve Signals are transmitted Simultaneously thre ug & This compresses ait the verons yéserwirs OF the abo helping +0 translocate blood out oF the abdomina | reservoirs tousards the hears Pr a resmy , Sruerease Grarrieres OF blood are made available For tne heart Pump CNS Ssunemfc Res ponse S when blood flouy to vac devreased stveyly enough putritional defittenay, to aust cerebral 'stremia 4 greatly increased lal wren of “02 ( (gene atkel Other adic subb tance) ~ \ The vasocunstvictor & Urdisauelerator neurons vic repenA direvtiy 4 become Strongly ergbea- tI of artertal pressure. hf) — 5 EXCISE, Yoga, ( Médi (Alio Plarasyrnpa ther é i) ysts heart rate HN) b ses (onducrion Wi) Lgey Cha tabi lity y Meuretrarumitter § acelylonoline Write down the ap af heart tale ( (entre VMC + in medulla, eS = je e€ e — ve GY Cardio aueletory Catdid inhibitory 4 { Regulation 1. Cardiac comers 5 Higher pie 2. 6Ffevent (Bain bridge ull 3 Baroy eueptors 1 Respratory ane, 4. Cherm orevepinrs ED a Pat b) Bareecormes (Bra 1\ rob \ Porte ALAR Sinus Sinus / po ER incveastd BP, decreased HR 2 Mayrey { lato C ayotd. inhibitory ) reeves the sq eal : 41 glossopha lang ear nerve e=> tarotid body p reuplos ry horkic body re) (hemor ecepiors G vagus nerve NS ——— ae ; 9 Reyponds hb Chang ¢ In Chemical! conth pues Seneca SEY! My porta. cecal f= { a Hy pevrcap nica itd AR — — (ai Guna ase ution. S (eardpacecterory ) ) | ce — #5) Hiaher “centers 3s Beals —_— — | Mc is conneuted with Hypetyalamus es —Limbie_Syslemn 9 relaltA with emobons, behaviour , — J e StL desire also contval VMC~ Cerebral cortex : Aven 3 - €mohen ~ Sympathe He —» ant. brady cardi, post - tracny & ada, : = es ak aa a =—@ ).| Guevues. FROM RespIRAUTEY enters ae ie Ed a — Pees inspiration - sed ear rate —2 Expiracron : used “heart tale aad a [Bain b bride Tele. —_- z VAC 4 cases uardip aueleratjon . — (Atria! wall “stuekehes), = —- “ta atyium . iy Aug Mrentecl Umpolar limb leads _Ruotd [SA rode + AV nade Bundle oF Hy = Rurkinjee bibers PAS 0 ig, Sha De ema pene Ricker it oo ce nC a one limb joined with sooo resitanu 3 leads — i) AVR = Right arm ; i) AVL > Vert arm iF > foot ( Augen } Unipolar dnist_ Una leade (entre (gad) Skernum V4 - 4 intercostal spare MSE HIgh Of the Slernum ¥2_- 4M intercostal Spare , jutk 1erk pf the sleroum = Vee hatway ple V2 4 Vy V4 5H snvercostal Spat, Mid claviculay Line ad 5h interwstal space, anterior axiliary line - mid ax) lary line Jpg RST] k SS See 2 | eel) a ste vf en. tlre i F e whe oe 5 y _rdepoarsation ay sven adap ° Slow sq na\ : delay nviniriwdar Malar iarion bee & ing SAT, —— Aapiinigtin 1 Twave ° Ges Comeltx Observations a. ji .| Rate COMM CC ee ‘ Rhythm = heque | P-voowe vol kage - _0.2MV |Muthods of réordirg gon, » seb hi ises ae thts Rvoaveg, (40) court squares. divide by 1500" - P-wancduration ~~. 9.06 seyord SAS | win 9-R nlerval QRS vortage ks duvatior | ST segment - 6. ae %- 4 |T-wonl ygltage (0 7 WANE Jurayon PRALTICAL CARDIAC GrFICVENCY TEST Harvard seep test : O-3mil | wight * 46 kg Basal pulse rate « \ if { ) 6010 oC ee 4o0 $00 go0 400 WI — gone] 200 03105/2% — HE _ paryi mins c nw tnst_pa in, bub etal ‘f ; 4 partent falls down Ave fo cage ep oqual Abformal WA Vatfe of cardia Oukp ab po ae he On | pic ci, heart ‘Dibtk iui peste ff) ae ; : ) ele -aaiele me SA node _danpaged Bee 2 Tsed heartrate isncphaa, 4 rsplration Av-nodal Sinoatrial block) : 7 rr oe ) See Cae: + Works ee ee a), P-wave absent 5 ) Psu Av node (damaged ee is due Wot. Tsch emia of Oy- ep 2. Inflammation of “py Node ) aay Compresston ee ‘ S [AV oF heart block ) (1) 4 ae ree i (11) 4 degree (partial block) 2 PR interval > 9-2 sec es ) » Signal 15 slightly slow asthe assed PRI with A 
