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HEENT - Nose Disorder Topics Chart

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2020/2021

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Disorder Cause Symptoms Diagnosis Treatment Notes
Acute Viral
Rhinitis
Contaminated objects
Airborne droplets
Rhinorrhea
Nasal congestion
Sneezing
Sore throat
Cough
Malaise HA
Fever possible (not common)
Exam: Nasal d/c,
edema/erythema of nasal
mucosa, pharyngitis possible
Symptomatic treatment
Fluids- hydration, warm fluids (soup, tea)
Nasal saline- flushes nasal cavity
Antipyretics/analgesics
Decongestants
Rhinovirus and
Coronavirus most
common
Acute Viral
Rhinosinusitis
Most common
Rhinovirus, , influenza virus,
parainfluenza virus
Numerous serologic types which is
why we stay susceptible
Patho: Viral inoculation via direct
contact to conjunctiva or nasal
mucosa > viral replication >
symptoms
Inflammation > hypersecretion
and increased vascular
permeability > transudation of
fluid
-
Impaired mucociliary clearance -
Risk factors: older age, smoking,
changes in atmospheric pressure,
swimming, asthma/allergies,
dental disease, immunodeficiency
Nasal congestion
Sinus pressure/pain (worse
with bending forward)
Watery to purulent
rhinorrhea
Erythema/edema of nasal
mucosa with discharge
Tenderness over sinuses
possible
Sneezing
Decreased sense of smell
Malaise
Sore throat (worse at night)
Headache
Cough (d/t post nasal drip)
Fever (maybe low grade)
Ear pressure (ET dysfxn)
ClinicalSelf limited
Symptomatic (nasal saline, NSAIDs,
decongest ants, nasal stero ids)
Rest & fluids
Last 10-14 days, first week worse
Acute: symptoms <4
weeks
Subacute: symptoms
4-12 weeks
Chronic: symptoms
persist >12 weeks
Recurrent acute:less
than 4 episodes/yr w
interim symptoms
resolution
Acute Bacterial
Rhinosinusitis
Uncommon
(0.5-2%)
Complication of viral infection
Maxillary is most common form
Streptoccoccus pneumonia (most
common), H. influenza, Moraxella
catarrhalis & group A strep
Patho: Mucosal inflammation >
ostial obstruction > negative
pressure > retained secretions >
secondary infections
Purulent nasal discharge
Nasal congestion
Facial/sinus pain/pressure
(edema)
Dental pain (can have with
viral too)
Erythema/edema of nasal
mucosa
Tenderness over sinuses
possible
Altered smell
Cough
Fever
Headache
Fatigue
Usually clinical
CT (contrast)- fluid in sinuses,
air bubbles
Culture (refractory or
complicated cases)
**typically distinguished from
viral by persistence of
symptoms for more then 10
days after onset or worsening
of symptoms within 1 0 days
after initial improvement
40-69% will improve within 2 wks without
abx
Symptomatic: NSAIDs, decongestants,
nasal ster oids
Consider abx when symptoms lasts longer
than 10-14 days or when symptoms are
severe or complicated by
immunodeficiency.
Adults 65 y/o 1st line:Am oxicillin
(500mg Q8H or 875mg Q12H) or
*Augmentin (875 mg Q12H)
Adults >65 y/o, hospitalization in
prior 5 days, abx use in the prior
month, immunocompromised,
multiple comorbidities or severe
infection 1st line:High dose
Augmentin (2000mg/125mg Q12H)
PCN allergy 1st line: Doxycycline or
(Clindamycin plus a cephalosporin)
Refer: failure to resolve after adequate
abx send to otolaryngologist for eval
Complications:
Preseptal/Orbital
Cellulitis
Subperiosteal
abscess
Osteomyelitis
Meningitis
Intracranial
abscess
Septic cavernous
sinus thrombosis
Allergic Rhinitis Seasonal-pollens and spores
Spring: Flowering shrub and tree
pollens
-
Summer: Flowering plants and
grasses
-
Fall: Ragweed and molds
-
Perennial-year round symptoms
Dust, house mites, air pollution
and pet dander
-
Patho: Allergens enter nose >
trigger IgE > IgE activates mast
cells > release of mediators
(histamine, prostaglandins) >
mediators stimulate blood vessels,
nerves, and glands > symptoms
-
Nasal
Itching
Watery rhinorrhea
Nasal Congestion
Sneezing
Ocular
Irritation
Pruritus
Conjunctival erythema
Excessive Tearing
Other
Fatigue
Poor sleep
*Similar to viral rhinitis but
usually persistent and may
show seasonal variation
External Exam
Allergic shiners (dark circles),
Dennie Morgan lines, Allergic
salute(transverse nasal crease)
Internal Exam
Pale bluish, edematous nasal
mucosa/turbinates due to
venous engorgement
Nasal polyps are associated
with long standing allergic
rhinitis.
Other Findings
Cobblestoning in the posterior
pharynx
Retracted TMs with or w/o
serous fluid behind them
Allergic conjunctivitis
Intranasal corticosteroids are 1st line
(fluticasone, triamcinolone)
Intranasal antihistamines (azelastine,
olopatadine)
Oral antihistamines (cetirizine, loratadine,
fexofenadine)
Cromolyn nasal spray
Montelukast (Singulair)-useful if
concomitant asthma or nasal polyposis
Avoid or reduce exposure to allergens
Refer: severe uncontrollable allergies can
be referred to allergist f or possible tes ting
and immunotherapy
Serum: RASTs, IgE immunoassays
Hypersensitivity skin testing
Additional Forms of
Rhinitis:
Vasomotor Rhinitis-
elderly, chronic
drip/runny nose due to
nerve
Rhinitismeticomentosa
-rebound congestion
(Afrin)
Nasal Polyps Can be seen in chronic sinusitis,
cystic fibrosis, allergies, and
asthma
Patho: Thought to be caused
mostly by chronic inflammation
Pale, edematous, mucosally
covered masses
Looks like water balloon
Nasal obstruction and
diminished smell
Intranasal corticosteroids 1st line
Short course of oral steroids
If large or meds unsuccessful they can be
removed surgically
*Avoid aspirin in patients with nasal
polyps and history of ast hma as it can
cause severe bronchospasm (ASA or
Samter’s tri ad)
Nasal Trauma Trauma Crepitance or palpably
mobile bony segments
Epistaxis, pain, black eyes
*r/o septal hematoma:
Septal cartilage re ceives its only
nutrition f rom its closely
Aimed at maintaining nasal airway
patency an d cosmetic appear ance
Closed reduction can be done under local
Nose & Sinus Disorders
Thursday, April 13, 2023 9:06 AM
Nose Disorders Page 1
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Disorder Cause Symptoms Diagnosis Treatment Notes Acute Viral Rhinitis

  • Contaminated objects
  • Airborne droplets
    • Rhinorrhea
    • Nasal congestion
    • Sneezing
    • Sore throat
    • Cough
    • Malaise HA
    • Fever possible (not common) Exam: Nasal d/c, edema/erythema of nasal mucosa, pharyngitis possible Symptomatic treatment
  • Fluids- hydration, warm fluids (soup, tea)
  • Nasal saline- flushes nasal cavity
  • Antipyretics/analgesics
  • Decongestants Rhinovirus and Coronavirus most common Acute Viral Rhinosinusitis Most common Rhinovirus, , influenza virus, parainfluenza virus

Numerous serologic types which is why we stay susceptible

Patho: Viral inoculation via direct contact to conjunctiva or nasal mucosa > viral replication > symptoms

Inflammation > hypersecretion and increased vascular permeability > transudation of fluid

  • Impaired mucociliary clearance Risk factors: older age, smoking, changes in atmospheric pressure, swimming, asthma/allergies, dental disease, immunodeficiency
  • Nasal congestion Sinus pressure/pain (worse with bending forward)

Watery to purulent rhinorrhea

Erythema/edema of nasal mucosa with discharge

Tenderness over sinuses possible

  • Sneezing
  • Decreased sense of smell
  • Malaise
  • Sore throat (worse at night)
  • Headache
  • Cough (d/t post nasal drip)
  • Fever (maybe low grade)
  • Ear pressure (ET dysfxn)
    • Clinical •Self limited Symptomatic (nasal saline, NSAIDs, decongestants, nasal steroids)
  • Rest & fluids
  • Last 10-14 days, first week worse Acute: symptoms < weeks

Subacute: symptoms 4-12 weeks

Chronic: symptoms persist >12 weeks

Recurrent acute: less than 4 episodes/yr w interim symptoms resolution

Acute Bacterial Rhinosinusitis Uncommon (0.5-2%)

  • Complication of viral infection
  • Maxillary is most common form Streptoccoccus pneumonia (most common), H. influenza, Moraxella catarrhalis & group A strep

Patho: Mucosal inflammation > ostial obstruction > negative pressure > retained secretions > secondary infections

  • Purulent nasal discharge
  • Nasal congestion Facial/sinus pain/pressure (edema)

Dental pain (can have with viral too)

Erythema/edema of nasal mucosa

Tenderness over sinuses possible

  • Altered smell
  • Cough
  • Fever
  • Headache
  • Fatigue
    • Usually clinical CT (contrast)- fluid in sinuses, air bubbles

Culture (refractory or complicated cases)

**typically distinguished from viral by persistence of symptoms for more then 10 days after onset or worsening of symptoms within 10 days after initial improvement 40-69% will improve within 2 wks without abx

Symptomatic: NSAIDs, decongestants, nasal steroids

Consider abx when symptoms lasts longer than 10-14 days or when symptoms are severe or complicated by immunodeficiency. Adults 65 y/o 1st line: Amoxicillin (500mg Q8H or 875mg Q12H) or *Augmentin (875 mg Q12H)

Adults >65 y/o, hospitalization in prior 5 days, abx use in the prior month, immunocompromised, multiple comorbidities or severe infection 1st line: High dose Augmentin (2000mg/125mg Q12H)

PCN allergy 1st line: Doxycycline or (Clindamycin plus a cephalosporin)

Refer: failure to resolve after adequate abx send to otolaryngologist for eval

Complications: Preseptal/Orbital Cellulitis

Subperiosteal abscess

  • Osteomyelitis
  • Meningitis Intracranial abscess

Septic cavernous sinus thrombosis

Allergic Rhinitis • Seasonal-pollens and spores Spring: Flowering shrub and tree pollens

Summer: Flowering plants and grasses

  • Fall: Ragweed and molds
  • Perennial-year round symptoms Dust, house mites, air pollution and pet dander

Patho: Allergens enter nose > trigger IgE > IgE activates mast cells > release of mediators (histamine, prostaglandins) > mediators stimulate blood vessels, nerves, and glands > symptoms

Nasal

  • Itching
  • Watery rhinorrhea
  • Nasal Congestion
  • Sneezing Ocular
  • Irritation
  • Pruritus
  • Conjunctival erythema
  • Excessive Tearing Other
  • Fatigue
  • Poor sleep *Similar to viral rhinitis but usually persistent and may show seasonal variation External Exam Allergic shiners (dark circles), Dennie Morgan lines, Allergic salute(transverse nasal crease)

Internal Exam Pale bluish, edematous nasal mucosa/turbinates due to venous engorgement

Nasal polyps are associated with long standing allergic rhinitis.

Other Findings Cobblestoning in the posterior pharynx

Retracted TMs with or w/o serous fluid behind them

  • Allergic conjunctivitis Intranasal corticosteroids are 1st line (fluticasone, triamcinolone)

Intranasal antihistamines (azelastine, olopatadine)

Oral antihistamines (cetirizine, loratadine, fexofenadine)

  • Cromolyn nasal spray Montelukast (Singulair)-useful if concomitant asthma or nasal polyposis
  • Avoid or reduce exposure to allergens Refer: severe uncontrollable allergies can be referred to allergist for possible testing and immunotherapy - Serum: RASTs, IgE immunoassays - Hypersensitivity skin testing

Additional Forms of Rhinitis: Vasomotor Rhinitis- elderly, chronic drip/runny nose due to nerve Rhinitismeticomentosa

  • rebound congestion (Afrin) Nasal Polyps Can be seen in chronic sinusitis, cystic fibrosis, allergies, and asthma

Patho: Thought to be caused mostly by chronic inflammation

Pale, edematous, mucosally covered masses

  • Looks like water balloon Nasal obstruction and diminished smell
  • Intranasal corticosteroids 1st line
  • Short course of oral steroids If large or meds unsuccessful they can be removed surgically

*Avoid aspirin in patients with nasal polyps and history of asthma as it can cause severe bronchospasm (ASA or Samter’s triad) Nasal Trauma Trauma Crepitance or palpably mobile bony segments

  • Epistaxis, pain, black eyes *r/o septal hematoma: Septal cartilage receives its only nutrition from its closely Aimed at maintaining nasal airway patency and cosmetic appearance
  • Closed reduction can be done under local

Nose & Sinus Disorders

Thursday, April 13, 2023 9:06 AM Nose Disorders Page 1

Epistaxis, pain, black eyes common

Make sure no palpable step off of the infraorbital rim

nutrition from its closely adherent mucoperichondrium. If untreated will result in loss of the nasal cartilage with resultant saddle nose deformity. Can become infected. Usually need to be drained. Refer. Closed reduction can be done under local or general anesthesia

  • Refer to ENT if septum is deviated Epistaxis Nasal trauma- nose picking, FB, forceful nose blowing
  • Rhinitis Dry nasal mucosa- low humidity, hot environment, or nasal O

• HTN

  • Bleeding disorders
  • Anticoagulated patients Hereditary hemorrhagic telangectosa (Osler Weber Rendu Syndrome)
  • Cocaine use
  • ETOH use Anterior - Most common (90-95%) - Kiesselbach plexus Posterior - 5-10% of cases Sphenopalatine artery branches or Woodruff plexus
  • More serious Important to consider underlying causes

Vital signs, mental status, airway, nasal exam

Lab assessment of bleeding may be indicated especially in recurrent cases (CB)

Refer: recurrent epistaxis, large volume epistaxis, episodic epistaxis with associated nasal obstruction, ongoing bleeding beyond 15-30 min, if you are not prepared to manage acute epistaxis Anterior 1st: Direct pressure on anterior 1/3 of nose continuously for 15 min in sitting position and slightly leaning forward

2nd: Short acting topical nasal decongestants (act as vasoconstrictor)

3rd: If bleeding does not subside and bleeding site visible can be cauterized with silver nitrate (most common), diathermy, or electrocautery

4th: If not visible can use hemostatic sealant, pneumatic nasal tamponade, anterior packing

Posterior

  • Consult an ENT Balloon catheters, foley catheter, cotton packing

Surgical ligation- used when packing fails to control life threatening hemorrhage

After control of epistaxis advise pt to AVOID: straining and vigorous exercise for several days

  • Sneezing
  • Spicy/hot foods Tobacco (may cause vasodilation)

If packing is going to remain in place for several days give: Antistaphylococcal abx (cephalexin, clindamycin) to reduce toxic shock syndrome

Nose Disorders Page 2