Physiotherapy notes 1, Study notes of Physiotherapy

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Typology: Study notes

2021/2022

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WEEK 1
THE MUSCULOSKELETAL EXAMINATION
- Most of orthopedic history involves pain
- Ask about:
- Location of pain,
- Hx of trauma,
- Localized vs diffuse,
- Acute vs chronic,
- Inflammatory vs noninflammatory,
- Limited ROM, systemic features,
- Always ask about previous orthopedic surgeries/internal hardware
- Monoarticular joint pain: one area of joint pain
- Polyarticular joint pain: multi-joint pain
- Timing of onset:
- Acute: developed over the course of hours
- Chronic: insidious, weeks-months
- Limited ROM: important to know if the ROM is stiffness or actually being stopped
- Tightness= perceived tightness or resistance to movement
- Systemic features: is there any fever, chills, rash, anorexia, weight loss, weakness
Joint & Systemic Disorders:
- Systemic Lupus Erythematosus: butterfly (malor) rash on cheeks
- Psoriatic arthritis: scaly rash, pitted, nails
- Lyme Disease
- Reiter’s Syndrome
- Gonococcal arthritis
Low back pain
- Always evaluate for bowel or bladder dysfunction
- DRE, test rectal sphincter
Neck pain
- Cervical spine important to evaluate
- Ask about radiation to arm or shoulder
- Numbness/tingling weakness?
Red flags for back pain/neck pain:
- Age >50yrs
- Hx of cancer
- Unexplained weight loss
- Pain lasting >1 month & not responding to Tx
- Night or rest pain
- History of IV drug use
- Presence of infxn
- As a general rule ALWAYS inspect the joint above and below an injury
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WEEK 1

THE MUSCULOSKELETAL EXAMINATION

- Most of orthopedic history involves pain - Ask about: - Location of pain, - Hx of trauma, - Localized vs diffuse, - Acute vs chronic, - Inflammatory vs noninflammatory, - Limited ROM, systemic features, - Always ask about previous orthopedic surgeries/internal hardware

  • Monoarticular joint pain: one area of joint pain
  • Polyarticular joint pain: multi-joint pain
  • Timing of onset:
    • Acute: developed over the course of hours
    • Chronic: insidious, weeks-months
  • Limited ROM: important to know if the ROM is stiffness or actually being stopped
    • Tightness= perceived tightness or resistance to movement
  • Systemic features: is there any fever, chills, rash, anorexia, weight loss, weakness

Joint & Systemic Disorders:

  • Systemic Lupus Erythematosus: butterfly (malor) rash on cheeks
  • Psoriatic arthritis: scaly rash, pitted, nails
  • Lyme Disease
  • Reiter’s Syndrome
  • Gonococcal arthritis

Low back pain

- Always evaluate for bowel or bladder dysfunction

  • DRE, test rectal sphincter

Neck pain

  • Cervical spine important to evaluate
  • Ask about radiation to arm or shoulder
  • Numbness/tingling weakness?

Red flags for back pain/neck pain :

  • Age >50yrs
  • Hx of cancer
  • Unexplained weight loss
  • Pain lasting >1 month & not responding to Tx
  • Night or rest pain
  • History of IV drug use
  • Presence of infxn
  • As a general rule ALWAYS inspect the joint above and below an injury

FRACTURE MANAGEMENT

  • Bone consists of cells imbedded within an abundant extracellular matrix of mineral and organic elements
  • Bone has the remarkable and unique ability to heal by complete regeneration rather than by scar tissue formation–fractures in bones initiate a continuous sequence of healing that includes: - Inflammation: the shorted phase of healing and begins immediately after injury - Repair: bone reparative process is stimulated by chemotactic factors released during inflammation - Remodeling: final phase of fracture healing begins approx 6 weeks after the injury
  • Age is one of the most important factors that influence bone healing:
    • Children's fractures heal rapidly
    • Fractures in adults heal slower

MANAGEMENT OF PEDIATRIC FRACTURES

  • In children’s fractures bone growth may be affected, abundant callus may form during healing, clinical healing is faster, fracture remodeling is more pronounced, children tolerate prolonged immobilization much better and rehabilitation after immobilization is usually not needed
  • Fractures in children are generally less complicated and are more often treated by closed means, nonunion is rare because of the abundant blood supply of growing bone

SHOULDER DISORDERS

SUBACROMIAL IMPINGEMENT SYNDROME (SHOULDER IMPINGEMENT)

- Subacromial impingement occurs in subacromial space between the lateral aspect of the acromion and the humeral head (shoulder blade rubbing against rotator cuff)

  • More of a chronic presentation not an acute injury
  • Causes: - MC caused by repetitive overhead activities
    • Rotator cuff tear
    • Wear and tear
    • Subacromial bone spurs

SIGNS/SYMPTOMS

  • Pain with overhead motion & internal rotation (putting on jacket, bra)
  • Nocturnal pain when sleeping on affected shoulder
  • Sense of instability
  • Things to know: hand dominance, occupation, hobbies
  • PE:
    • Inspection typically normal
    • Palpation may have tenderness over the area - Positive Neer Test & Hawkins test

DIAGNOSIS

  • Clinical presentation
  • XRAYs:
    • AP scapula view
    • Lateral scapular view
    • Axillary lateral view
    • AP acromioclavicular view
  • MRI:
    • (if suspecting ligament tear) Superior for tendon/ligament injuries

TREATMENT

  • Activity modification
  • Physical therapy
  • NASIDs/ice–short term only
  • Corticosteroid injection
  • Surgical intervention

ROTATOR CUFF TEARS

  • Tear of one of the shoulder muscles
  • Rotator cuff is made of SITS: - Supraspinatus, infraspinatus, tere minor, subscapularis
  • Epidemiology:
    • 28% of pts >60 yrs have full thickness tear
    • 65% of patients > 70 yrs have full thickness tear

SIGNS/SYMPTOMS

  • Weakness or pain with overhead movement
  • Difficulty lifting arm with limited active ROM
  • Nocturnal pain - Similar findings with impingement syndrome but more pronounced
  • PE:
    • Positive Neer & Hawkins
    • Weakness with resisted strength testing → full thickness tear

DIAGNOSIS

- XRAY: - High grade acromial spurs–high incidence of tears - Acromioclavicular joint arthritis–mimics cuff tears - Acromioclavicular Joint spurs can cause tears

  • MRI : best for visualizing tears
  • MRA (form of MRI): show partial or small tears
  • Ultrasound for those who cannot undergo MRI

TREATMENT

- Partial rotator cuff tears - Conservative therapy - Physical therapy with strengthening of scapular/rotator cuff muscle - Best for sedentary patients - ~40% progress to full thickness tear in 2 yrs - Full thickness tears - Do not heal well with conservative therapy & tend to worsen

SIGNS/SYMPTOMS

  • Dull, achy shoulder pain
  • Sharp pain with movement
  • Preceding trauma
  • Crepitus (clicking or popping)
  • PE:
    • No single maneuver that can confirm the diagnosis (inside the joint)
    • Complete shoulder examination including assessment of neurovascular status
    • May have limited ROM due to pain
    • Tenderness to palpation

DIAGNOSIS

**- Clinical presentation

  • MRI**

TREATMENT

  • NSAIDS or acetaminophen
  • Rest
  • Physical therapy
  • Surgical (recovery time 6-12 mos)

BICEP TENDONITIS

- Inflammation of the long head of the biceps brachii

  • CauseS:
    • Associated with impingement, rotator cuff tears, stenosis of biceps groove
    • Athletes
    • Repetitive overhead movements such as carpenters, electricians, and mechanical wheelchair users

SIGNS/SYMPTOMS

- Progressively worsening pain over the anterior shoulder

  • Pain may radiate from the anterior shoulder down the anterior arm
  • PE:
    • Positive Speeds test
    • Positive Yergason's test

DIAGNOSIS

  • Clinical presentation
  • XRAYs: important in identifying other causes of pain such as fractures and arthritis
  • MRI: frequently used to assist in Dx & may demonstrate thickening and increased signal within the tendon
  • Ultrasound: may reveal presence of an effusion or absence of the biceps tendon in the intertubercular groove

TREATMENT

  • NSAIDs, rest, PT
  • Corticosteroid injections if no improvement in 6-8 weeks
  • Surgical release–only for refractory cases

SIGNS/SYMPTOMS

  • Acute injury
  • Pain
  • Swelling
  • Shoulder deformity
    • Collar bone appears to be ‘sticking up’
  • PE:
    • Tenderness over the AC joint
    • Passive cross-body adduction of the arm elicits pain

DIAGNOSIS

  • Clinical presentation
  • XRAYs may be negative (Type I)

TREATMENT

- Type I & II: NSAIDs, rest, ice (+/- sling)

  • Type III : controversial– conservative vs surgical treatment
  • Type IV-VI : surgical intervention

SHOULDER DISLOCATION

- MC commonly dislocated joint

  • Age at time of initial dislocation is important risk factor for recurrent dislocations
  • Anterior
  • Posterior
  • Sternoclavicular subluxation/dislocation

ANTERIOR DISLOCATION

**- Traumatic abduction & external rotation

  • MC**

DIAGNOSIS

- Clinical presentation & XRAY

  • Neurovascular status MUST be assessed before and after reduction of the shoulder

COMPLICATIONS

TREATMENT

  • Conservative non-operative treatment
  • NSAIDS and PT

ADHESIVE CAPSULITIS–FROZEN SHOULDER

  • Contracture of glenohumeral capsule leading to severe shoulder stiffness and pain
  • Women > men
  • Typically 40-65 yrs old
  • Self-limiting but debilitating condition
  • Associated with:
    • Trauma after chest/breast surgery
    • Prolonged immobilization
    • Endocrine disorders
      • Diabetes
      • Thyroid disorder

SIGNS/SYMPTOMS

  • Phase I: inflammatory
    • Painful
    • No clinical findings
    • 4-6 mos
  • Phase II: freezing
    • Stiffness
    • Less pain
    • 4-6 mos
  • Phase III: thawing
    • Regains ROM slowly approx 1 yr
    • Course is delayed with endocrine disorders

DIAGNOSIS

  • Clinical presentation
  • XRAY to rule out glenohumeral arthritis

TREATMENT

  • NSAIDs, physical therapy
  • Possible corticosteroid injection
  • Oral prednisone

INTERVENTIONS

  • Manipulation under anesthesia
  • Arthroscopy for lysis of adhesions

COMPLICATIONS

  • Non-union or malunion
  • Degenerative changes

HUMERAL FRACTURES

SIGNS/SYMPTOMS

  • Recent trauma/fall
  • Pain that increases with movement
  • Swelling
  • Deformity

NEER CLASSIFICATION

  • The Neer system divides the proximal humerus into four parts and considers not the fracture line but the displacement as being significant in terms of classification. The four parts are the humeral head, the great tuberosity the lesser tuberosity and the humeral shaft

Main complication: avascular necrosis

HUMERAL FRACTURES–SHAFT

- Midshaft humeral fracture: - Can injure radial nerve - Radial nerve runs in a spiral groove of humerus - Causes wrist drop–consult if any neuro deficit

TREATMENT

  • Non-operative– functional brace if minimal angulation or <3cm shortening
  • Operative
    • Open fracture floating elbow
    • Polytrauma
    • Pathological fracture
    • Associated brachial plexus injury–neuro deficit warrants?

UPPER EXTREMITY DISORDERS

OVERVIEW

OPEN VS CLOSED FRACTURES

  • Most open fractures are obvious because bone is visible–either protruding through the skin or within a wound
  • Some open fractures are more subtle–when there is any wound in the same area as a fracture it is assumed to be an **open fracture
  • Treatment–** antibiotics, consult ortho, wash out thoroughly may need to go to OR - Patient needs to see ortho ASAP
  • Nerves to be aware of in the elbow and forearm (stem from C5-T1):
  • Know blood supply of arm
  • Know olecranon bursa anatomy

ELBOW FRACTURES

  • Supracondylar fracture
  • Condyle fracture
    • Single column
    • Two column distal humerus fracture
    • Capitellum fracture
    • Olecranon fracture

FAT PADS AND ELBOW FRACTURES

- Ability to lock elbow in full extension - Shown to be ~95% accurate in excluding a fracture

  • Point tenderness–helps to locate area for imaging
  • Most abnormal fat pads–intraarticular fracture with blood in joint
  • Small anterior fat pad–can be normal
  • Larger anterior fat pad–abnormal
  • Posterior fat pad–virtually always abnormal