Pathophysiology Part 2; WGU D236, Exams of Advanced Education

Pathophysiology Part 2; WGU D236

Typology: Exams

2025/2026

Available from 02/11/2026

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Pathophysiology Part 2; WGU D236
RAAS system
renin-angiotensin-aldosterone system
1. increases vasoconstriction
2. increases blood volume
3. increases blood pressure
4. increases Na+
isotonic
Describes a solution whose solute concentration is equal to the solute concentration inside a cell
Hypotonic
Having a lower concentration of solute than another solution
Hypertonic
Having a higher concentration of solute than another solution.
Hyponatremia
deficient sodium in the blood
- causes seizures, coma, neuro damage, brain swelling
- slow fluid replacement
hypernatremia
high sodium in the blood
- tachycardia, irritability, flushed skin
- diuresis or slow fluid replacement
hypokalemia
deficient potassium in the blood
- cardiac arrhythmias, anorexia, absent reflexes
hyperkalemia
excessive potassium in the blood
- mental confusion, apathy, numb extremities
hypocalcemia
deficient calcium in the blood
- Chvostek's sign
- Trousseau's sign
- seizures
hypercalcemia
excessive calcium in the blood
- muscle flaccidity, bone tenderness, constipation
Hypophosphatemia
A serum phosphorus level less than 2.7 mg/dL.
- tremors
- osteomalacia
- hyporeflexia
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Pathophysiology Part 2; WGU D

RAAS system renin-angiotensin-aldosterone system

  1. increases vasoconstriction
  2. increases blood volume
  3. increases blood pressure
  4. increases Na+ isotonic Describes a solution whose solute concentration is equal to the solute concentration inside a cell Hypotonic Having a lower concentration of solute than another solution Hypertonic Having a higher concentration of solute than another solution. Hyponatremia deficient sodium in the blood
  • causes seizures, coma, neuro damage, brain swelling
  • slow fluid replacement hypernatremia high sodium in the blood
  • tachycardia, irritability, flushed skin
  • diuresis or slow fluid replacement hypokalemia deficient potassium in the blood
  • cardiac arrhythmias, anorexia, absent reflexes hyperkalemia excessive potassium in the blood
  • mental confusion, apathy, numb extremities hypocalcemia deficient calcium in the blood
  • Chvostek's sign
  • Trousseau's sign
  • seizures hypercalcemia excessive calcium in the blood
  • muscle flaccidity, bone tenderness, constipation Hypophosphatemia A serum phosphorus level less than 2.7 mg/dL.
  • tremors
  • osteomalacia
  • hyporeflexia

Hyperphosphatemia a serum phosphorus level that exceeds 4.5 mg/dL

  • symptoms related to hypercalcemia Hypomagnesemia deficient magnesium in the blood
  • Babinski's sign
  • Tetany
  • Cardiac arrhythmias hypermagnesemia an excess of magnesium in the blood plasma
  • hypotension
  • cardiac arrest
  • sedation Blood pH range 7.35-7. PCO2 range 35-45 mmHg PO2 range 90-100 mmHg HCO3- range 22-26 mEq/L SaO2 range 95-100% hypo/hypercapnia low/high carbon dioxide hypo/hyperoxia low/high oxygen metabolic acidosis decreased pH, decreased CO metabolic alkalosis increased pH, Increased CO Respiratory acidosis decreased pH, increased CO Respiratory alkalosis increased pH, decreased CO What is the most common cause of metabolic alkalosis? vomiting, diarrhea, gastric suctioning Familial dilated cardiomyopathy Hereditary disease causing enlarged chambers
  • decreased heart pumping abilities leading to heart failure type 1 diabetes Diabetes of a form that usually develops during childhood or adolescence and is characterized by a severe deficiency of insulin, leading to high blood glucose levels.
  • Genetic predisposition
  • Little to no insulin production

HPT axis Hypothalmic-pituitary-thyroid axis

  • More everyday action salpingitis inflammation of the fallopian tubes (or eustachian tubes) BUN blood urea nitrogen 10-20 mg/dL